April 2010
Volume 51, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2010
Effect of Dietary Fish Oil Upon the Development of Murine Herpetic Retinal Necrosis
Author Affiliations & Notes
  • I. Berra
    Ocular Laboratory, Biofundus, Buenos Aires, Argentina
    Pathology, Laboratorio Investigaciones Oculares, Universidad de Buenos Aires, Argentina
  • G. Zapata
    Pathology, Laboratorio Investigaciones Oculares, Universidad de Buenos Aires, Argentina
  • A. Berra
    Pathology, Laboratorio Investigaciones Oculares, Universidad de Buenos Aires, Argentina
  • Footnotes
    Commercial Relationships  I. Berra, None; G. Zapata, None; A. Berra, None.
  • Footnotes
    Support  UBACYT 403 / PICT2007-2252
Investigative Ophthalmology & Visual Science April 2010, Vol.51, 2906. doi:
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      I. Berra, G. Zapata, A. Berra; Effect of Dietary Fish Oil Upon the Development of Murine Herpetic Retinal Necrosis. Invest. Ophthalmol. Vis. Sci. 2010;51(13):2906.

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Abstract
 
Purpose:
 

To examine the effects of diets enriched with n-3 and n-6 polyunsaturatedfatty acids (PUFAs) in a murine model of herpetic chorioretinitis.

 
Methods:
 

BALB/c mice were fed on high-fat diets which contained Menhadenoil (rich in n-3 PUFAs) (diet 1), Safflower oil (rich in n-6PUFAs) (diet 2), or Corn oil as control(diet 3), 14 days previouslyand 12 days following anterior chamber (AC) HSV-1 inoculation.After 14 days of treatment and before infection, splenic cellproliferation was evaluated as well as cytokines synthesis onculture supernatants.Development of chorioretinitis and encephalitiswas followed-up by clinical and histological signs. On day 12p.i. mice were sacrificed. Both eyes and brain were obtainedfor viral titration and RT-PCR analysis.

 
Results:
 

Mice fed on Menhaden oil experimented a decreased synthesisof TNF-α, IFN-γ, IL-2 and IL-10 in splenic cells isolated andstimulated by Con-A. Post HSV-1 inoculation, mice fed on Menhadenoil showed clinical and histological signs of chorioretinitisin both eyes and encephalitis. Moreover, high titers of viruswere recovered in ocular and brain tissue homogenates.

 
Conclusions:
 

Our findings indicate that mice fed on fish oil have impairedTh-1 immune respons which could lead to increased virus titersand consequent development of encephalitis and chorioretinisin both eyes. We also conclude that small but detectable amountsof virus (1x10(4) PFU/ml) could be present at uninoculated eyeswithout clinical and histopathological evidence of retinitis.It suggests that the triggering of retinal injury should requirea critical threshold of virus.  

 

 
Keywords: uveitis-clinical/animal model • pathology: experimental • inflammation 
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