April 2010
Volume 51, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2010
Effects of Anti-VEGF Treatment on Retinal Vasculature in Retinal Vein Occlusion as Determined by Ultra-Wide Field Fluorescein Angiography
Author Affiliations & Notes
  • J. J. Tseng
    Ophthalmology, Vitreous Retina Macula Consultants NY, New York, New York
    Ophthalmology, Columbia University, New York, New York
  • S. Kandula
    Ophthalmology, Vitreous Retina Macula Consultants NY, New York, New York
  • L. K. Chang
    Ophthalmology, Columbia University, New York, New York
  • K. Freund
    Ophthalmology, Vitreous Retina Macula Consultants NY, New York, New York
  • Footnotes
    Commercial Relationships  J.J. Tseng, None; S. Kandula, None; L.K. Chang, None; K. Freund, None.
  • Footnotes
    Support  The Macula Foundation
Investigative Ophthalmology & Visual Science April 2010, Vol.51, 3577. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      J. J. Tseng, S. Kandula, L. K. Chang, K. Freund; Effects of Anti-VEGF Treatment on Retinal Vasculature in Retinal Vein Occlusion as Determined by Ultra-Wide Field Fluorescein Angiography. Invest. Ophthalmol. Vis. Sci. 2010;51(13):3577.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose: : To investigate the effect of inhibition of anti-vascular endothelial growth factor (VEGF) therapy for macular edema in the setting of retinal vein occlusion (RVO) on central and peripheral retinal circulation.

Methods: : Two patients with macular edema, one with central retinal vein occlusion (CRVO) and one with branch retinal vein occlusion (BRVO) underwent ultra-wide field (200-degree) photography and fluorescein angiography (FA) at the time of diagnosis. The patient with CRVO received an intravitreal injection of ranibizumab (0.5 mg/0.05 ml). The patient with BRVO received an intravitreal injection of bevacizumab (1.25mg/0.05 ml). Both eyes were reevaluated with ultra-wide field photography and FA at 1 week and 5 weeks post-treatment. Photographs and FA were analyzed for post-treatment changes in arteriovenous (AV) transit time, vessel caliber, vessel leakage, peripheral capillary nonperfusion, retinal hemorrhages, and nerve fiber layer infarcts.

Results: : In both eyes, there were no changes in AV transit time or peripheral capillary nonperfusion at 1 and 5 weeks after anti-VEGF therapy. Decreased vessel caliber and reduction in intraretinal hemorrhages and nerve fiber layer (NFL) infarcts were observed in both eyes at the 5-week examination. In the patient with CRVO, a recurrence of vascular leakage was noted at 5 weeks.

Conclusions: : Anti-VEGF therapy in the setting of RVO may initially decrease vessel caliber, retinal hemorrhages, and NFL infarcts but does not appear to affect AV transit time or areas of peripheral capillary nonperfusion. The beneficial effects of anti-VEGF therapy in RVO appear transient, and multiple injections may be required for control of macular edema.

Keywords: vascular occlusion/vascular occlusive disease • edema • vascular endothelial growth factor 
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×