April 2010
Volume 51, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2010
Epidermal Growth Factor, a Potent Initiator of RPE Cell Proliferation
Author Affiliations & Notes
  • K. Steindl
    Ludwig Boltzmann Institute for Retinology & Biomicroscopic Lasersurgery, Department of Ophthalmology, Rudolf Foundation Clinic, Vienna, Austria
  • M. E. Boulton
    Department of Anatomy and Cell Biology, University of Florida, Florida, Florida
  • W. Krugluger
    Department of Clinical Chemistry, Donauspital, Vienna, Austria
  • P. Haas
    Ophthalmology,
    Rudolf Foundation Clinic, Vienna, Austria
  • A. Dossenbach-Glaninger
    Department of Clinical Chemistry,
    Rudolf Foundation Clinic, Vienna, Austria
  • S. Binder
    Ophthalmology,
    Rudolf Foundation Clinic, Vienna, Austria
  • Footnotes
    Commercial Relationships  K. Steindl, None; M.E. Boulton, None; W. Krugluger, None; P. Haas, None; A. Dossenbach-Glaninger, None; S. Binder, None.
  • Footnotes
    Support  None.
Investigative Ophthalmology & Visual Science April 2010, Vol.51, 5962. doi:
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    • Get Citation

      K. Steindl, M. E. Boulton, W. Krugluger, P. Haas, A. Dossenbach-Glaninger, S. Binder; Epidermal Growth Factor, a Potent Initiator of RPE Cell Proliferation. Invest. Ophthalmol. Vis. Sci. 2010;51(13):5962.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : To investigate the regulatory effect of EGF on cell proliferation vs. differentiation in ARPE19 cells after different incubation periodes (24h vs. 48h) for obtaining ideal conditions for feasible rejuvenation and autologous transplantation of RPE cells.

Methods: : To evaluate gene expression of RPE-specific differentiation and proliferation markers as well as transcriptional and translational activity of ß-Catenin signaling markers by FACS and RT-PCR after 24h and 48h EGF treatment.

Results: : Our data showed a significant decrease of RPE 65, CRALBP and Cytokeratin 18 in ARPE-19 cells even after 24h EGF treatment. In addition an increase of Cyclin D1 expression and a significant decrease of GSK-3ß and ß-Catenin were equally observed after 24h and 48h EGF treatment. Cell cycle studies revealed an increase of ARPE cells in S-G2/M phase after 24h EGF treatment.

Conclusions: : Our recently published data identified EGF as a potent initiator of RPE proliferation via activation of the ß-Catenin Signalling pathway after 48h treatment. Our new data demonstrate the induction of proliferation and upregulation of the ß-Catenin signaling pathway by EGF even after 24 hours incubation. As ideal cell culture conditions are essential to maintain RPE-specific phenotype, short incubation times enhance RPE cell quality for feasible rejuvenation and subsequent autologous transplantation of RPE cells.

Keywords: retinal pigment epithelium • age-related macular degeneration • aging 
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