April 2010
Volume 51, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2010
Potential Role of Mitogen-Activated Protein Kinase Phosphatase 1 in AMD
Author Affiliations & Notes
  • S. Dridi
    Ophthalmology & Visual Sciences, University of Kentucky, Lexington, Kentucky
  • H. Kaneko
    Ophthalmology & Visual Sciences, University of Kentucky, Lexington, Kentucky
  • M. Kleinman
    Ophthalmology & Visual Sciences, University of Kentucky, Lexington, Kentucky
  • R. Albuquerque
    Ophthalmology & Visual Sciences, University of Kentucky, Lexington, Kentucky
  • J. Baffi
    Ophthalmology & Visual Sciences, University of Kentucky, Lexington, Kentucky
  • J. Ambati
    Ophthalmology & Visual Sciences, University of Kentucky, Lexington, Kentucky
  • Footnotes
    Commercial Relationships  S. Dridi, None; H. Kaneko, None; M. Kleinman, None; R. Albuquerque, None; J. Baffi, None; J. Ambati, Genentech, Allergan, Quark, C; University of Kentucky, P.
  • Footnotes
    Support  NIH/NEI, Doris Duke Charitable Foundation, Burroughs Wellcome Fund, RPB
Investigative Ophthalmology & Visual Science April 2010, Vol.51, 6174. doi:
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    • Get Citation

      S. Dridi, H. Kaneko, M. Kleinman, R. Albuquerque, J. Baffi, J. Ambati; Potential Role of Mitogen-Activated Protein Kinase Phosphatase 1 in AMD. Invest. Ophthalmol. Vis. Sci. 2010;51(13):6174.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : AMD is a leading cause of visual impairment. Inflammation and apoptotic cell death of RPE and photoreceptors are well recognized to be components of AMD, however the underlying intracellular signaling pathways are not completely understood. MKP1, which negatively regulates MAPK signaling, play an essential function in inflammation and apoptosis. Here we investigated the potential role of MKP1 in AMD.

Methods: : Real-time PCR and Western Blot were used to determine MKP1 mRNA and protein levels in RPE cells from AMD and normal subjects and in old and young mice. Immunofluorescence was used to determine the localization of MKP1 in human RPE cells cultured in normal conditions.

Results: : MKP1 is ubiquitously expressed in the human eye with high levels in retina and RPE. It is mainly localized in the cytoplasm in normal conditions and it is downregulated in RPE of old mice compared to that of young counterparts and in human RPE from AMD compared to that from normal subjects.

Conclusions: : These data suggest a potential role of MKP1 in AMD disease.

Keywords: age-related macular degeneration • aging • gene/expression 
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