April 2010
Volume 51, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2010
Glycosaminoglycan Distribution in the Human Macula
Author Affiliations & Notes
  • T. D. Keenan
    Faculty of Medical and Human Sciences,
    University of Manchester, Manchester, United Kingdom
  • S. J. Clark
    Wellcome Trust Centre for Cell-Matrix Research, Faculty of Life Sciences,
    University of Manchester, Manchester, United Kingdom
  • R. J. Baldwin
    Stem Cell Glycobiology Group, Materials Science Centre,
    University of Manchester, Manchester, United Kingdom
  • T. H. van Kuppevelt
    Department of Biochemistry, Radboud University Nijmegen Medical Center, Nijmegen, The Netherlands
  • A. J. Day
    Wellcome Trust Centre for Cell-Matrix Research, Faculty of Life Sciences,
    University of Manchester, Manchester, United Kingdom
  • P. N. Bishop
    Wellcome Trust Centre for Cell-Matrix Research, Faculty of Life Sciences,
    University of Manchester, Manchester, United Kingdom
  • Footnotes
    Commercial Relationships  T.D. Keenan, None; S.J. Clark, None; R.J. Baldwin, None; T.H. van Kuppevelt, None; A.J. Day, None; P.N. Bishop, None.
  • Footnotes
    Support  Macular Disease Society (no grant code) and National Institute for Health Research (no grant code)
Investigative Ophthalmology & Visual Science April 2010, Vol.51, 6320. doi:
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      T. D. Keenan, S. J. Clark, R. J. Baldwin, T. H. van Kuppevelt, A. J. Day, P. N. Bishop; Glycosaminoglycan Distribution in the Human Macula. Invest. Ophthalmol. Vis. Sci. 2010;51(13):6320.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : To investigate the localization of glycosaminoglycans (GAGs) in the human macula using immunohistochemistry and fluorescence microscopy.

Methods: : Postmortem human eye tissue was obtained, following removal of the cornea for transplantation, from consenting eye donors. Macular tissue was fixed in 4% paraformaldehyde prior to freezing, and serial sections were cut. These were probed with a series of antibodies raised against GAG chains; primary antibodies were detected using fluorescently labelled secondary antibodies. In some cases, tissue sections were digested with GAG-degrading enzymes prior to application of the primary antibodies.

Results: : GAG chains demonstrated differential localization within human macular tissue. Within the neurosensory retina, chondroitin sulfate (CS) was seen predominantly in the interphotoreceptor matrix, and dermatan sulfate (DS) mainly in the ganglion cell layer. Heparan sulfate (HS) and hyaluronan (HA) were observed throughout the neurosensory retina, and HS displayed segregation by sulfation pattern. Minimal staining for keratan sulfate (KS) was found in the neurosensory retina. In general, GAGs with low levels of sulfation (including HA) were observed at the internal limiting membrane. HS, DS and KS were present to varying degrees in the retinal pigment epithelium, Bruch's membrane and in the walls of choroidal blood vessels.

Conclusions: : GAG chains display differential localization in the human macula according to both GAG chain type and degree of sulfation. This has important implications for understanding eye development and disease, and for planning novel therapeutic strategies.

Keywords: immunohistochemistry • proteoglycans/glycosaminoglycans • macula/fovea 
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