May 2007
Volume 48, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2007
Intravitreal Bevacizumab as an Adjunct to Ovine Hyaluronidase (Vitrase®) in the Long-Term Management of Vitreous Hemorrhage
Author Affiliations & Notes
  • A. Toor
    Ophthalmology, Mount Sinai School of Medicine, New York, New York
    Ophthalmology, Elmhurst Hospital Center, New York, New York
  • D. Chruscicki
    Ophthalmology, Mount Sinai School of Medicine, New York, New York
    Ophthalmology, Elmhurst Hospital Center, New York, New York
  • A. Macedo
    Ophthalmology, Mount Sinai School of Medicine, New York, New York
    Ophthalmology, Elmhurst Hospital Center, New York, New York
  • R. L. Lieberman
    Ophthalmology, Mount Sinai School of Medicine, New York, New York
    Ophthalmology, Elmhurst Hospital Center, New York, New York
  • R. M. Fischer
    Ophthalmology, Mount Sinai School of Medicine, New York, New York
    Ophthalmology, Elmhurst Hospital Center, New York, New York
  • Footnotes
    Commercial Relationships A. Toor, None; D. Chruscicki, None; A. Macedo, None; R.L. Lieberman, None; R.M. Fischer, None.
  • Footnotes
    Support None.
Investigative Ophthalmology & Visual Science May 2007, Vol.48, 265. doi:
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    • Get Citation

      A. Toor, D. Chruscicki, A. Macedo, R. L. Lieberman, R. M. Fischer; Intravitreal Bevacizumab as an Adjunct to Ovine Hyaluronidase (Vitrase®) in the Long-Term Management of Vitreous Hemorrhage. Invest. Ophthalmol. Vis. Sci. 2007;48(13):265.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose:: Intravitreal ovine hyaluronidase (Vitrase®) is a novel therapy believed to expedite the resorption of vitreous hemorrhage by causing the dissolution of collagen and hyaluronic complexes in vitreous humor. Bevacizumab (Avastin, Genentech), a monoclonal antibody that targets vascular endothelial growth factor (VEGF), has been shown to cause regression of retinal neovascularization . We present a long-term follow up of patients with vitreous hemorrhage treated with a combination of intravitreal hyaluronidase followed by intravitreal bevacizumab.

Methods:: A retrospective chart review was done of patients with established VH secondary to proliferative diabetic retinopathy (PDR) or retinal vein occlusion, who were then treated with a single intravitreal injection of Vitrase® (55 IU/0.05ml), followed by intravitreal bevacizumab (1.25mg/0.05ml). Data collected included patient demographics, best corrected visual acuity (BCVA) with VH, at 2wks, 1mth, 2mths, 3mths, 4mths, 5mths and 6mths post-injection, treatment with panretinal photocoagulation (PRP) post-injection and fluorescein angiogram (FA) results.

Results:: 10 eyes from 9 patients (5M, 4F), age 52+8.4yrs, were included. Initial BCVA ranged from 20/60 to LP. After Vitrase® injection, 5 eyes had a ≥3 line gain in BVCA, 1 had <3 line gain, and 4 eyes showed no change in BCVA. Intravitreal bevacizumab was injected in all eyes at a median 8wks post-Vitrase for persistent VH, inability to place sufficient PRP, or for vitreous rebleeds due to persistent retinal neovascularization. In eyes with a ≥3 line gain in BVCA post-Vitrase® (n=5), retinal neovascularization regressed on FA, vision improved further by ≥3 lines and 680+211 additional PRP burns were placed post-bevacizumab. In eyes with <3 line gain in BCVA post-Vitrase® (n=5), bevacizumab injection did not improve vision or clear VH further in 4 out of 5 eyes. Poor response to Vitrase®/bevacizumab was associated with vision worse than CF on presentation.

Conclusions:: Intravitreal bevacizumab can be a useful adjunct in improving vision, facilitating placement of PRP and promoting regression of neovascularization in VH when there is an initial response to Vitrase®. In eyes with VH that fails to clear post-Vitrase®, adjunctive bevacizumab is unlikely to improve visual outcome. A larger prospective protocol is required to confirm the findings of this pilot study.

Keywords: diabetic retinopathy • injection • vitreous 
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