May 2006
Volume 47, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2006
LIM–Homeodomain Transcription Factor Lim1 (Lhx1) Regulates the Migration of Retinal Horizontal Cells
Author Affiliations & Notes
  • R.A. Poche
    Program in Developmental Biology, Baylor College of Medicine, Houston, TX
  • M.A. Raven
    Neuroscience Research Institute and Department of Psychology, University of California at Santa Barbara, Santa Barbara, CA
  • K.M. Kwan
    Department of Molecular Genetics, University of Texas M.D. Anderson Cancer Center, Houston, TX
  • Y. Furuta
    Department of Biochemistry and Molecular Biology, University of Texas M.D. Anderson Cancer, Houston, TX
  • B.E. Reese
    Neuroscience Research Institute and Department of Psychology, University of California at Santa Barbara, Santa Barbara, CA
  • R.R. Behringer
    Department of Molecular Genetics, University of Texas M.D. Anderson Cancer Center, Houston, TX
  • Footnotes
    Commercial Relationships  R.A. Poche, None; M.A. Raven, None; K.M. Kwan, None; Y. Furuta, None; B.E. Reese, None; R.R. Behringer, None.
  • Footnotes
    Support  EY07102 and EY11087
Investigative Ophthalmology & Visual Science May 2006, Vol.47, 2776. doi:
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      R.A. Poche, M.A. Raven, K.M. Kwan, Y. Furuta, B.E. Reese, R.R. Behringer; LIM–Homeodomain Transcription Factor Lim1 (Lhx1) Regulates the Migration of Retinal Horizontal Cells . Invest. Ophthalmol. Vis. Sci. 2006;47(13):2776.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : Homeodomain transcription factors are known to have essential roles in retinal cell–fate determination and subsequent differentiation. It has been proposed that these transcription factors function, in a combinatorial code, to pattern the seven retinal cell classes derived from a common progenitor pool. To further define the importance of a combinatorial transcription factor network to retinal development, we are characterizing the role of the homeodomain transcription factor Lim1 (Lhx1). Beginning at embryonic day 15.5 and continuing into adulthood, Lim1 is expressed in horizontal cells of the mouse retina. The spatial and temporal expression of Lim1 suggests that Lim1 may have a role in regulating horizontal cell precursor migration to the mature horizontal cell layer.

Methods: : In order to circumvent the embryonic lethality of the Lim1 null mutant, mice homozygous for a Lim1 floxed allele were crossed to transgenic mice expressing a retina–specific Cre recombinase thereby generating retina–specific knockouts.

Results: : Conditional knockout retinas show a change in cell positioning whereby cells, normally fated to become horizontal cells, are ectopically localized to the inner aspects of the inner nuclear layer, many being positioned at the border of the inner plexiform layer. Previous studies in chick suggest that horizontal cells migrate bi–directionally, from the ventricular zone then to the vitreal side of the neuroepithelium, before migrating back again to their final laminar position in the inner nuclear layer adjacent to the outer plexiform layer. In the conditional mutant retinas, Lim1–/– horizontal cells are apparently incapable of initiating or completing the second phase of migration to the prospective horizontal cell layer and remain "stuck" amongst the amacrine cells where they continue to differentiate, extending their processes basally into the inner plexiform layer. Thus, the new local environment is possibly influencing the ectopic cell morphology.

Conclusions: : Based on the normal expression of Lim1 throughout the process of horizontal cell migration, and on the conditional knockout phenotype, we propose that Lim1 regulates the second phase of horizontal cell bi–directional migration.

Keywords: horizontal cells • transcription factors • retinal connections, networks, circuitry 
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