May 2006
Volume 47, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2006
Inhibition of Experimental Proliferative Vitreoretinopathy by Gene Therapy With PTEN
Author Affiliations & Notes
  • Y.–H. Chen
    Ophthalmology, Chang Gung memorial hospital, Kaohsiung, Taiwan Republic of China
  • H.–K. Kuo
    Ophthalmology, Chang Gung memorial hospital, Kaohsiung, Taiwan Republic of China
  • P.–C. Wu
    Ophthalmology, Chang Gung memorial hospital, Kaohsiung, Taiwan Republic of China
  • L.–S. Cheng
    Ophthalmology, Chang Gung memorial hospital, Kaohsiung, Taiwan Republic of China
  • M.–H. Tai
    Laboratory of Genetic Engineering, Veterans General hospital, Kaohsiung, Taiwan Republic of China
  • Footnotes
    Commercial Relationships  Y. Chen, None; H. Kuo, None; P. Wu, None; L. Cheng, None; M. Tai, None.
  • Footnotes
    Support  National Science Council Taiwan
Investigative Ophthalmology & Visual Science May 2006, Vol.47, 4666. doi:
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      Y.–H. Chen, H.–K. Kuo, P.–C. Wu, L.–S. Cheng, M.–H. Tai; Inhibition of Experimental Proliferative Vitreoretinopathy by Gene Therapy With PTEN . Invest. Ophthalmol. Vis. Sci. 2006;47(13):4666.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : To evaluate the inhibitory activity of PTEN on the formation of experimental PVR

Methods: : In vitro, the RPE cells were infected with adenovirus encoding PTEN. The cell proliferation was determined at one and two days using MTT assay. The PTEN gene expression was analyzed by Western blot. In vivo, the left eye of pigmented rabbits was injected with 1×105 cells of RPE transfected with PTEN. In contrast, the right eye of rabbits was injected with 1×105 cells of RPE transfected with GFP only.

Results: : The MTT analysis revealed the proliferation rate was decreased to the extent of 80% in the Ad–PTEN group compared with the control group. Western blot analysis confirmed the exogenous PTEN and GFP gene were successfully introduced to the RPE cell by the adenoviruses. PVR progression was similar in both groups of rabbits during the 4–week follow–up. At day 28, the mean PVR grade was 2.3 in the experimental group and 1.7 in the control group. Two groups didn’t have the statistical difference about the PVR grading.

Conclusions: : The PTEN gene is successfully transinfected to the RPE cell, but the suppressive effect was relative limited. The exogenous PTEN gene did not modify the development of cell–induced PVR.

Keywords: proliferative vitreoretinopathy • gene transfer/gene therapy • retinal pigment epithelium 
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