May 2005
Volume 46, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2005
Virulence Contribution of Pseudomonas Aeruginosa Elastase and Alkaline Protease in a Pseudomonas Putida Model of Rabbit Keratitis
Author Affiliations & Notes
  • B.A. Thibodeaux
    Microbiology/Immunology, LSU Health Sciences Center, New Orleans, LA
  • A.R. Caballero
    Microbiology/Immunology, LSU Health Sciences Center, New Orleans, LA
  • M.E. Marquart
    Microbiology/Immunology, LSU Health Sciences Center, New Orleans, LA
  • J. Tommassen
    Department of Molecular Microbiology, Utrecht University, Utrecht, Netherlands Antilles
  • A. Lazdunski
    Laboratoire de Chimie Bacterienne, Centre National de la Recherche Scientifique, Marseille, France
  • R.J. O'Callaghan
    Microbiology/Immunology, LSU Health Sciences Center, New Orleans, LA
  • Footnotes
    Commercial Relationships  B.A. Thibodeaux, None; A.R. Caballero, None; M.E. Marquart, None; J. Tommassen, None; A. Lazdunski, None; R.J. O'Callaghan, None.
  • Footnotes
    Support  NEI grant EY12961
Investigative Ophthalmology & Visual Science May 2005, Vol.46, 1168. doi:
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      B.A. Thibodeaux, A.R. Caballero, M.E. Marquart, J. Tommassen, A. Lazdunski, R.J. O'Callaghan; Virulence Contribution of Pseudomonas Aeruginosa Elastase and Alkaline Protease in a Pseudomonas Putida Model of Rabbit Keratitis . Invest. Ophthalmol. Vis. Sci. 2005;46(13):1168.

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Abstract
 
Abstract:
 

To measure the specific virulence contributions of two Pseudomonas aeruginosa proteases, elastase B and alkaline protease, when expressed separately by Pseudomonas putida in a rabbit model of bacterial keratitis.

 

P. putida KT2660 was transformed with one of two plasmid constructs encoding either elastase B (lasB) or alkaline protease (aprA) along with the accessory proteins required for its secretion (aprD, aprE, aprF, and aprI). Protease expression was confirmed by zymography and Western blotting. P. putida expressing elastase B (Put:plasBT), alkaline protease (Put:pAP), or vector alone (Put:p20) was injected intrastromally (103 colony forming units [CFU] in 10 µl) into rabbit corneas (n=6). Infected corneas were graded by slit lamp examination (SLE) at 20, 24, 28, and 32 hours (h) post infection (PI). Rabbits were sacrificed at 32 h PI and the log CFU (± SEM) per cornea were determined.

 

SLE scores for each group at 20, 24, 28, and 32 h PI are shown in the table below.

 

 

SLE scores for corneas infected with Put:plasBT were significantly higher than those infected with vector alone at all time points (p ≤ 0.008). SLE scores for corneas infected with Put:pAP were not significantly higher than those infected with vector alone at any time point (p ≥ 0.1). At both 24 and 28 h PI, the SLE scores for corneas infected with Put:lasBT were significantly higher than those infected with Put:pAP (p ≤ 0.002). At 32 h PI, the mean log CFU for corneas infected with Put:lasBT (5.51 ± 0.48) was significantly lower (p ≤ 0.02) than those infected with Put:p20 (6.80 ± 0.13) or Put:pAP (6.66 ± 0.12).

 

The expression of elastase B, but not alkaline protease, in the P. putida model of keratitis significantly increased SLE scores at all time points measured. This suggests that elastase B contributes to the pathology observed during P. aeruginosa keratitis.

 

 
Keywords: Pseudomonas • keratitis • pathology: experimental 
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