May 2005
Volume 46, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2005
Glial Cell Line–derived Neurotrophic Factor in the Vitreous of Patients With Proliferative Diabetic Retinopathy
Author Affiliations & Notes
  • N. Nishikiori
    Department of Ophthalmology, Sapporo Medical University School of Medicine, Sapporo, Japan
  • A. Tashimo
    Department of Ophthalmology, Sapporo Medical University School of Medicine, Sapporo, Japan
  • Y. Mitamura
    Department of Ophthalmology, Sapporo Medical University School of Medicine, Sapporo, Japan
  • K. Ohtsuka
    Department of Ophthalmology, Sapporo Medical University School of Medicine, Sapporo, Japan
  • Footnotes
    Commercial Relationships  N. Nishikiori, None; A. Tashimo, None; Y. Mitamura, None; K. Ohtsuka, None.
  • Footnotes
    Support  None.
Investigative Ophthalmology & Visual Science May 2005, Vol.46, 3103. doi:
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      N. Nishikiori, A. Tashimo, Y. Mitamura, K. Ohtsuka; Glial Cell Line–derived Neurotrophic Factor in the Vitreous of Patients With Proliferative Diabetic Retinopathy . Invest. Ophthalmol. Vis. Sci. 2005;46(13):3103.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose: Glial cell line–derived neurotrophic factor (GDNF) is a member of the neurotrophic factor family. Previous studies indicated the implication of GDNF in glial development. We have reported that expression levels of GDNF receptor family α–component were significantly higher in the epiretinal membrane of proliferative diabetic retinopathy (PDR). Theses studies suggest a possibility that GDNF receptors may be involved in the formation of PDR epiretinal membrane. Here, we attempted to determine whether intravitreous GDNF levels are elevated in PDR. Methods: We assayed GDNF levels in vitreous samples from 39 consecutive patients with PDR (22 eyes) and macular hole (controls, 17 eyes) that underwent pars plana vitrectomy. Undiluted vitreous samples were collected during the vitrectomy before intraocular infusion. The vitreous samples were centrifuged, and the supernatants were stored at –80°C until the assay was performed. Vitreous GDNF concentrations were measured using an enzyme–linked immunosorbent assay. Results: Vitreous GDNF levels were 136.3±216.5 pg/ml (mean±SD) in PDR and 8.0±17.1 pg/ml in the controls. GDNF levels were significantly higher in PDR than in the controls (P=0.011531). Conclusions: This result indicates increased intravitreous GDNF levels in PDR eyes, suggesting that GDNF may play a role in the pathogenesis of PDR. CR: None

Keywords: vitreous • cytokines/chemokines • diabetic retinopathy 
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