May 2005
Volume 46, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2005
Presumed Toxoplasmic Unifocal Solitary Choroiditis
Author Affiliations & Notes
  • L. Oliveira
    Universidade de São Paulo, Ribeirão Preto, Brazil
    Universidade Federal de Uberlandia, Uberlândia, Brazil
  • M.A. Ferreira
    Universidade Federal de Uberlandia, Uberlândia, Brazil
  • J.D. O. Pena
    Universidade Federal de Uberlandia, Uberlândia, Brazil
  • C.T. Espinhosa
    Universidade Federal de Uberlandia, Uberlândia, Brazil
  • N.M. Silva
    Universidade Federal de Uberlandia, Uberlândia, Brazil
  • R. Jorge
    Universidade de São Paulo, Ribeirão Preto, Brazil
  • Footnotes
    Commercial Relationships  L. Oliveira, None; M.A. Ferreira, None; J.D.O. Pena, None; C.T. Espinhosa, None; N.M. Silva, None; R. Jorge, None.
  • Footnotes
    Support  None.
Investigative Ophthalmology & Visual Science May 2005, Vol.46, 5063. doi:
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    • Get Citation

      L. Oliveira, M.A. Ferreira, J.D. O. Pena, C.T. Espinhosa, N.M. Silva, R. Jorge; Presumed Toxoplasmic Unifocal Solitary Choroiditis . Invest. Ophthalmol. Vis. Sci. 2005;46(13):5063.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose: To report the clinical features of the unifocal choroiditis establishing evidences of presumed toxoplasmic etiology. Methods:Interventional study where 7 healthy young patients (among 20 and 30 years old), experienced acute unilateral visual loss secondary to unifocal choroiditis. Clinical features, natural course, and follow–up were established. Results:All patients exhibited a solitary, elevated, yellow–white active focus of choroiditis with overlying subretinal fluid and in some cases subretinal hemorrhage. The lesions measured approximately 1 disc diameter size, with none or mild vitreous inflamation. Vision loss was related to the location of the lesion or the subretinal fluid with respect to the fovea. The patients with prolonged follow–up, elevated white plaque lesions persisted overtime. One out of 7 patients presented previous history of uveitis or corioretinal inactive scars. All of them presented the serum IgG positive for toxoplasmosis. All other systemic evaluations were unremarkable. Two patients were submitted to intravitreal tap, where protein chain reaction (PCR) for diferent etiologies were being analized. Conclusions: To our knowledge, this is the first report of a series of patients with diagnostic of Unifocal Solitary Choroiditis, where all of them presented laboratory evidence of previous toxoplasmic infection.

Keywords: choroid • toxoplasmosis • uveitis-clinical/animal model 
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