May 2004
Volume 45, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2004
Role of the outer microvessel layer venules in the early complications of experimental BRVO : an in vivo study
Author Affiliations & Notes
  • M. Paques
    Fondation Ophtalmologique Rothschild and INSERM U 592, Paris, France
  • M. Simonutti
    INSERM U 592, Paris, France
  • J.–A. Sahel
    Fondation Ophtalmologique Rothschild and INSERM U 592, Paris, France
  • Footnotes
    Commercial Relationships  M. Paques, None; M. Simonutti, None; J. Sahel, None.
  • Footnotes
    Support  none
Investigative Ophthalmology & Visual Science May 2004, Vol.45, 456. doi:
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      M. Paques, M. Simonutti, J.–A. Sahel; Role of the outer microvessel layer venules in the early complications of experimental BRVO : an in vivo study . Invest. Ophthalmol. Vis. Sci. 2004;45(13):456.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose: To analyze the microvascular consequences of experimental branch retinal vein occlusion (BRVO). Methods: Following laser–induced BRVO in pigmented rats, funduscopy, fluorescein and indocyanine green angiography were monitored with a confocal scanning laser ophthalmoscope. Results: Immediately following BRVO, blood–retinal barrier rupture, perivenular opacification, ICG parietal staining and collateral circulation formation were observed. The blood–retinal barrier rupture and the collateral circulation involved mainly the venules of the outer microvessel layer as demonstrated by serial optical sectioning. BRB rupture appeared as a combination of diffuse leakage through the deep venule wall and pericapillary fluid collections. Conclusions: The early funduscopic and angiographic alterations following BRVO involve mainly the venules of the outer microvessel layer. These alteration may initiate chronic microvascular remodeling and blood–retina barrier rupture.

Keywords: vascular occlusion/vascular occlusive disease • imaging methods (CT, FA, ICG, MRI, OCT, RTA, SLO, ultrasound) 
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