May 2004
Volume 45, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2004
Analysis of Counterreceptors of Galectins–3 and –7 in Mouse Corneal Epithelial Cells
Author Affiliations & Notes
  • Z. Cao
    New England Eye Center, Dept. of Ophthalmology and Center for Vision Research, Tufts University School of Medicine, Boston, MA
  • F.–T. Liu
    Department of Dermatology, University of California Davis School of Medicine, Davis, CA
  • N. Panjwani
    New England Eye Center, Dept. of Ophthalmology and Center for Vision Research, Tufts University School of Medicine, Boston, MA
  • Footnotes
    Commercial Relationships  Z. Cao, None; F. Liu, None; N. Panjwani, None.
  • Footnotes
    Support  NIH EY07088, P30 13078 and Massachusetts Lions Eye Research Fund
Investigative Ophthalmology & Visual Science May 2004, Vol.45, 1163. doi:
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      Z. Cao, F.–T. Liu, N. Panjwani; Analysis of Counterreceptors of Galectins–3 and –7 in Mouse Corneal Epithelial Cells . Invest. Ophthalmol. Vis. Sci. 2004;45(13):1163.

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Abstract

Abstract: : Purpose: We have previously shown that carbohydrate–binding proteins, galectins–3 and –7, are expressed in corneal epithelium and that the exogenous addition of these galectins stimulate the re–epithelialization of corneal wounds in a mouse animal model (J. Biol. Chem. 277:42299–42305, 2002). Since, most of the extracelluar matrix molecules, growth factors and growth factor receptors are glycosylated, it is logical to hypothesize that one or more of these molecules serve as counterreceptors of galectins and, that the galectins influence re–epithelialization of corneal wound by binding to and influencing the function of the specific counterreceptors. The goal of the present study was to identify the counterreceptors of galectins–3 and –7 and to determine whether the two galectins bind to similar or distinct counterreceptors. Methods: Primary cultures of corneal epithelium were prepared using corneas of 6– to 12–month old mice (C57BL/6 and 129 mixed genetic background). Membrane extracts of corneal epithelial cell cultures were electrophoresed on SDS–polyacrylamide gels and protein blots were probed with biotinylated galectins–3– and –7 in the presence and absence of ß–lactose. In some experiments, the membrane extracts were chromatograhed on galectin affinity columns, the bound material was eluted by ß–lactose and analyzed by lectin blot analysis using biotinylated galectins. Results: Galectin–3 and –7 bound to distinct glycoprotein counterreceptors. Galectin–3 bound to two major (116–kD and 120–kD) and two minor components (74–kD and 64–kD). In contrast, galectin–7 bound to four major (60–kD, 52–kD, 47–kD and 35–kD) and a number of minor components. In each case, binding of the galectins to the glycoproteins was abolished by ß–lactose. Conclusions: Galectins–3 and –7 bind to distinct counterreceptors. Studies are underway to establish the identity of these receptors. This study should help understand the molecular mechanism by which galectins influence re–epithelialization of corneal wounds.

Keywords: cell adhesions/cell junctions • glycoconjugates/glycoproteins • cornea: epithelium 
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