May 2004
Volume 45, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2004
Protective effect of donepezil against glutamate neurotoxicity in rat retinal ganglion cells
Author Affiliations & Notes
  • A. Miki
    Ophthalmology, Osaka University, Suita, Japan
  • Y. Otori
    Ophthalmology, Osaka University, Suita, Japan
  • M. Okada
    Ophthalmology, Osaka University, Suita, Japan
  • Y. Tano
    Ophthalmology, Osaka University, Suita, Japan
  • Footnotes
    Commercial Relationships  A. Miki, None; Y. Otori, None; M. Okada, None; Y. Tano, None.
  • Footnotes
    Support  none
Investigative Ophthalmology & Visual Science May 2004, Vol.45, 846. doi:
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      A. Miki, Y. Otori, M. Okada, Y. Tano; Protective effect of donepezil against glutamate neurotoxicity in rat retinal ganglion cells . Invest. Ophthalmol. Vis. Sci. 2004;45(13):846.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose: To examine the protective effect of donepezil, an acetylcholinesterase inhibitor which was developed as a therapeutic agent for Alzheimer’s disease, in cultured retinal ganglion cells (RGCs). Methods: RGCs were purified from dissociated rat retinal cells (postnatal day 6–8), using a modified two–step panning method and cultured in serum–free medium containing neurotrophic factors and forskolin. Survival of RGCs after exposure to glutamate (Glu, 25µM), with or without donepezil (Dnp, 100nM–100µM) was measured by calcein–acetoxymethyl ester staining after 3 days of exposure. Surviving RGCs were defined as cells with a calcein–stained cell body and a process extending at least two cell diameters from the cell body. Results: The application of Glu caused RGC death. Simultaneous application of Dnp reduced glutamate–induced RGC death in a dose–dependent manner. (Survival ratio: Glu only vs. Glu+Dnp (100µM), 61.7±9.4% vs. 91.7±14.5%, mean±SD n=9, p<0.001) Conclusions: These findings suggest that donepezil may inhibit glutamate–induced cell death in purified RGCs.

Keywords: ganglion cells • retinal culture • neuroprotection 
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