May 2004
Volume 45, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2004
PHOTORECEPTOR DEGENERATION INDUCES AN INCREASE IN Rac1 EXPRESSION
Author Affiliations & Notes
  • D. Hamassaki–Britto
    Dept Histology & Embryology, Univ Sao Paulo, Sao Paulo, Brazil
  • M.A. Belmonte
    Dept Histology & Embryology, Univ Sao Paulo, Sao Paulo, Brazil
  • L.M. Castro
    Dept Histology & Embryology, Univ Sao Paulo, Sao Paulo, Brazil
  • M.F. Santos
    Dept Histology & Embryology, Univ Sao Paulo, Sao Paulo, Brazil
  • Footnotes
    Commercial Relationships  D. Hamassaki–Britto, None; M.A. Belmonte, None; L.M. Castro, None; M.F. Santos, None.
  • Footnotes
    Support  FAPESP, CNPq, PRONEX–MCT (Brazil)
Investigative Ophthalmology & Visual Science May 2004, Vol.45, 3613. doi:
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      D. Hamassaki–Britto, M.A. Belmonte, L.M. Castro, M.F. Santos; PHOTORECEPTOR DEGENERATION INDUCES AN INCREASE IN Rac1 EXPRESSION . Invest. Ophthalmol. Vis. Sci. 2004;45(13):3613.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose:To investigate the expression of the Rho GTPases Rac1 and Cdc42 and their effectors p21–activated kinases 1–4 (PAKs) in two models of retinal degeneration: light–induced and rd mice. Methods:Adult Balb/c mice dark–adapted for 18h were light–stimulated (5000 lux) for 2h, and immediately sacrificed or kept in the dark for additional 6, 14 or 24 h. Rd mice (C3H/HeJ) were sacrificed at postnatal days 9, 11 or 15 days. The eyes were enucleated and processed for immunofluorescence. Apoptosis was evaluated using TUNEL. Results:Although not visible in the control, Rac1 expression in the outer nuclear layer (ONL) became progressively evident after light stimulation, reaching its maximum after 24 h. Similar observation was made in rd mice: the number of Rac1–positive cells in the ONL was higher in 11 days–old, when compared to 9 days–old animals. From 15 days–old on, Rac1 labeling became reduced. Cdc42 was not expressed in Balb/c and rd photoreceptors. In Balb/c mice, only PAKs 3 and 4 were expressed in photoreceptors. PAK 3 was present in the outer segments, whereas PAK4 was uniformly distributed throughout the ONL. Photoreceptor degeneration apparently did not alter the distribution of these proteins. Conclusions:The results suggest a role for Rac1 during photoreceptor degeneration in mice, in a pathway that might involve PAKs 3 and 4.

Keywords: retinal degenerations: cell biology • photoreceptors • cell death/apoptosis 
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