May 2003
Volume 44, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2003
Age-Related Posttranslational Modifications of Crystallins in Rat Retina
Author Affiliations & Notes
  • D.A. Ferrington
    Ophthalmology, U of M - Twin Cities, Minneapolis, MN, United States
  • R.J. Kapphahn
    Ophthalmology, U of M - Twin Cities, Minneapolis, MN, United States
  • C.M. Ethen
    Biochemistry, Molecular Biology, Biophysics, U of M - Twin Cities, Minneapolis, MN, United States
  • L. Higgins
    Mass Spectrometry Laboratory, U of M - Twin Cities, Minneapolis, MN, United States
  • E.A. Peters
    Mass Spectrometry Laboratory, U of M - Twin Cities, Minneapolis, MN, United States
  • Footnotes
    Commercial Relationships  D.A. Ferrington, None; R.J. Kapphahn, None; C.M. Ethen, None; L. Higgins, None; E.A. Peters, None.
  • Footnotes
    Support  NIA Grant RO3AG19024, Research to Prevent Blindness, AFAR, Foundation Fighting Blindness
Investigative Ophthalmology & Visual Science May 2003, Vol.44, 1601. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      D.A. Ferrington, R.J. Kapphahn, C.M. Ethen, L. Higgins, E.A. Peters; Age-Related Posttranslational Modifications of Crystallins in Rat Retina . Invest. Ophthalmol. Vis. Sci. 2003;44(13):1601.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Abstract: : Purpose: Crystallins are small heat shock proteins with chaperone-like function that prevent heat and oxidative stress-induced aggregation of misfolded proteins. It has been reported that crystallins undergo age-related posttranslational modifications in the lens. These modifications can disrupt the conformation of the protein and alter chaperone function. Here we describe for the first time posttranslational modifications of crystallins in the rat retina. Methods: Retinal homogenates from young and old F344BN rats were analyzed for posttranslational modifications using 2D gel electrophoresis and immunochemical detection of phosphoserine, acetylated lysine, and 4-hydroxy-2-nonenal adducts on proteins. Protein identification was achieved by enzymatic digestion followed by MALDI-TOF mass spectrometry of extracted peptides. Protein identification was confirmed by MS/MS sequencing of peptides. Results: In this study, we identified posttranslational modifications in alpha A, alpha B, and beta B2 crystallin, including oxidation, phosphorylation, and acetylation. For each crystallin, one major spot and many additional spots with altered migration, indicative of changes in intrinsic charge and/or mass from that of the major spot, were observed. Spots with the same mass, but altered charge migrated as more acidic species. The chemical modifications observed here alter the net charge of the protein, which accounts for the appearance of these acidic species. Truncated forms were observed as spots with altered mass. Age-related differences were observed for each crystallin in the number of spots with altered migration as well as the type of posttranslational modification detected. Conclusions: In the rat retina, alpha A, alpha B, and beta B2 crystallin exhibit increased age-related posttranslational modifications. The increased amount of chemically modified crystallins suggests there is less protection from stress-induced protein aggregation in aged retina.

Keywords: aging • protein modifications-post translational • retina 
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×