May 2003
Volume 44, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2003
Retinoid Affinity Biotinylation Reveals a Retinoid Binding Role For Rpe65
Author Affiliations & Notes
  • R.R. Rando
    Bio Chemistry & Mol Pharm, Harvard Medical School, Boston, MA, United States
  • W. Jahng
    Bio Chemistry & Mol Pharm, Harvard Medical School, Boston, MA, United States
  • C. David
    Bio Chemistry & Mol Pharm, Harvard Medical School, Boston, MA, United States
  • N. Nesnas
    Department of Chemistry, Columbia University, New York, NY, United States
  • K. Nakanishi
    Department of Chemistry, Columbia University, New York, NY, United States
  • Footnotes
    Commercial Relationships  R.R. Rando, None; W. Jahng, None; C. David, None; N. Nesnas, None; K. Nakanishi, None.
  • Footnotes
    Support  EY04096
Investigative Ophthalmology & Visual Science May 2003, Vol.44, 3508. doi:
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      R.R. Rando, W. Jahng, C. David, N. Nesnas, K. Nakanishi; Retinoid Affinity Biotinylation Reveals a Retinoid Binding Role For Rpe65 . Invest. Ophthalmol. Vis. Sci. 2003;44(13):3508.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose: RPE65 is a major membrane protein of the retinal pigment epithelium (1,2). While knockout studies in mice suggest an important role for this protein in the operation of the visual cycle in vivo (3), a biochemical function for this protein remains obscure. This study addresses this issue. Methods: In the current studies, RPE65 is specifically and covalently labeled by the retinyl ester analog (3R)- 3-[boc-lys(biotinyl)-O]-all trans-retinol chloroacetate 1 at low µM concentrations to explore the retinoid binding capacity of RPE65. Results: The affinity labeling of RPE65 by 1 is blocked by added retinoids, including analog 2. The stoichiometry of labeling is two, and two cysteine residues (C231 and C448) of RPE65 are specifically labeled by 1. The known homology of RPE65 to ß-carotene-15, 15'-dioxygenase may be relevant here, because a ß-carotene binding site contains the elements of two retinoid binding sites. Conclusions: These studies demonstrate that RPE65 is a retinoid binding protein. Absent any demonstrated enzymatic activity for RPE65, a plausible function for this protein may be to act as a retinyl ester binding protein. Retinyl ester binding proteins would mobilize these highly hydrophobic retinoids, making them available for further processing in the visual cycle. 1. Bavik, C. O., Busch, C., Eriksson, U. (1992) J. Biol. Chem. 267, 23035-23042. 2. Hamel, C. P., Tsilou, E., Pfeffer, B. A., Hooks, J. J., Detrick, B., Redmond, T. M. (1993) J. Biol. Chem. 268, 15751-15757. 3. Redmond, T. M., Yu, S., Lee, E., Bok, D., Hamasaki, D., Chen, N., Goletz, P., Ma, J.-X., Crouch, R., Pfeifer, K. (1998) Nature Genet. 20, 344-351.

Keywords: retinoids/retinoid binding proteins • retinal pigment epithelium 
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