May 2003
Volume 44, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2003
Genetic Susceptibility to Development of Severe Forms of Diabetic Retinopathy. Possible Mechanisms of Interactions with Hypertension
Author Affiliations & Notes
  • J.M. Lopes De Faria
    Ophthalmology, University of Campinas, Campinas, SP, Brazil
  • T.C. Correa
    Ophthalmology, University of Campinas, Campinas, SP, Brazil
  • A.V. Ferrari
    Ophthalmology, University of Campinas, Campinas, SP, Brazil
  • J.B. Lopes de Faria
    Internal Medicine, University of Campinas, Campinas, SP, Brazil
  • Footnotes
    Commercial Relationships  J.M. Lopes De Faria, None; T.C. Correa, None; A.V. Ferrari, None; J.B. Lopes de Faria, None.
Investigative Ophthalmology & Visual Science May 2003, Vol.44, 3904. doi:
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      J.M. Lopes De Faria, T.C. Correa, A.V. Ferrari, J.B. Lopes de Faria; Genetic Susceptibility to Development of Severe Forms of Diabetic Retinopathy. Possible Mechanisms of Interactions with Hypertension . Invest. Ophthalmol. Vis. Sci. 2003;44(13):3904.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose: To investigate the effects of DM on neuro retinal cell replication and on vascular permeability in SRH and their normotensive control WKY rats. Methods: Diabetes was induced by an intravenous injection of streptozotocin in SHR and WKY with 4 and 12 weeks old.After 2 weeks of DM, the animals were sacrificed,the eyes enucleated, fixed in paraformaldehyde and embedded in paraffin. The retinas were used for BrDU immunohystochemical or for protein extraction for p27 Western blot assays.To evaluate retina vascular permeability, a quantitative method using Evans Blue was performed. Results: Neural cell proliferation was lower in diabetic SHR than in control SHR and in diabetic and control WKY rats (0 vs 0.031vs 0.042vs 0.042, respectively, p=0.001) with 4 and 12 weeks of age (0.025vs 1.18vs 0.091vs 0.149, respectively, p=0.001). The p27 retinal proteins levels were higher in SHR after induction of DM. There was a breakdown of retinal permeability in diabetic rats, mainly in SHR. Conclusion: DM inhibites retinal neural cell proliferation, and the hypertension amplifies this effect, possible by increasing the p27 levels in the retina. The vascular permeability may be influenced by the neuro retinal elements.

Keywords: diabetes • diabetic retinopathy • ganglion cells 
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