December 2002
Volume 43, Issue 13
Free
ARVO Annual Meeting Abstract  |   December 2002
Evaluation of a Novel Photosensitizer MV6401 as a Photodynamic Therapy Agent using a Primate Choroidal Neovasularization Model
Author Affiliations & Notes
  • TA Ciulla
    Department of Ophthalmology Indiana Univ Sch of Medicine Indianapolis IN
  • MH Criswell
    Department of Ophthalmology Indiana Univ Sch of Medicine Indianapolis IN
  • RP Danis
    Department of Ophthalmology Indiana Univ Sch of Medicine Indianapolis IN
  • WJ Snyder
    Miravant Medical Technologies Santa Barbara CA
  • W Small
    Miravant Medical Technologies Santa Barbara CA
  • Footnotes
    Commercial Relationships    T.A. Ciulla, Miravant Medical Technologies F; M.H. Criswell, Miravant Medical Technologies F; R.P. Danis, Miravant Medical Technologies F; W.J. Snyder, Miravant Medical Technologies E; W. Small, Miravant Medical Technologies E. Grant Identification: NIH grant EY1119-01, Miravant Medical Technologies Inc.; and Research to Prevent Blindness, Inc.
Investigative Ophthalmology & Visual Science December 2002, Vol.43, 614. doi:
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      TA Ciulla, MH Criswell, RP Danis, WJ Snyder, W Small; Evaluation of a Novel Photosensitizer MV6401 as a Photodynamic Therapy Agent using a Primate Choroidal Neovasularization Model . Invest. Ophthalmol. Vis. Sci. 2002;43(13):614.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose: Photodynamic therapy (PDT) is an evolving technology for treating choroidal neovascularization (CNV) in maculopathies, particularly age-related macular degeneration (AMD). The effectiveness of novel PDT photosensitizer MV6401 (Miravant Pharmaceuticals, Inc.) to limit CNV was evaluated in the squirrel monkey CNV model. Methods: To induce experimental CNV, an IRIS diode laser (532 nm, 0.05 sec duration, 650 mW, 75 µm) established a 3x3 macular grid of photocoagulation sites (PS) in 8 squirrel monkey eyes. On post-laser day 28, animals were IV injected with MV6401 (0.15 µmols/kg) which exhibits an absorption peak at 659 nm. At post-injection times (from 20-120 minutes) the nasal and central PS columns were selectively treated with laser light at the drug's activation wavelength (664 nm, 500 mW/cm2 irradiance, 800 µm spot dia), delivered at duration-dependent light doses of 10 and 20 J/cm2 respectively. Results: On day 35, moderate to complete closure of CNV and choriocapillaris vessels (accompanied by negligible to low tissue damage) was apparent around PS receiving PDT treatments 60-80 minutes post-injection, including sites where extensive neovascularization had developed. Greater retinal damage was observed at sites treated prior to 60 minutes post-injection, whereas decreased vessel closure corresponded to post-injection treatment times longer than 80 minutes. Paradoxically, less damage was noted at central macular sites receiving 20 J/cm2 light dosages, compared to nasal sites receiving only 10 J/cm2, possibly reflecting variations in vascular density across the macula. Control sites exhibited non-obstructed CNV with RBC's. Conclusion: Photosensitizer MV6401 demonstrates effective CNV closure; a post-injection delay is necessary to achieve optimal treatment results. Refinement of light dosage parameters within different portions of the macula deserves further study.

Keywords: 308 age-related macular degeneration • 346 choroid: neovascularization • 516 photodynamic therapy 
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