December 2002
Volume 43, Issue 13
Free
ARVO Annual Meeting Abstract  |   December 2002
Autosomal Dominant Macular Dystrophy: Evidence for a Novel Disease-causing Gene
Author Affiliations & Notes
  • ST O'Connor
    Cole Eye Institute Cleveland Clinic Foundation Cleveland OH
  • Z Yang
    Cole Eye Institute Cleveland Clinic Foundation Cleveland OH
  • S Thirumalaichary
    Cole Eye Institute Cleveland Clinic Foundation Cleveland OH
  • C Trallis
    Cole Eye Institute Cleveland Clinic Foundation Cleveland OH
  • K Zhang
    Cole Eye Institute Cleveland Clinic Foundation Cleveland OH
  • Footnotes
    Commercial Relationships   S.T. O'Connor, None; Z. Yang, None; S. Thirumalaichary, None; C. Trallis, None; K. Zhang, None. Grant Identification: Cole Eye Institute, Steinbach Fund, NEI, Fight For Sight
Investigative Ophthalmology & Visual Science December 2002, Vol.43, 810. doi:
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    • Get Citation

      ST O'Connor, Z Yang, S Thirumalaichary, C Trallis, K Zhang; Autosomal Dominant Macular Dystrophy: Evidence for a Novel Disease-causing Gene . Invest. Ophthalmol. Vis. Sci. 2002;43(13):810.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose: To report a kindred with an autosomal dominantly inherited macular dystrophy without linkage to or associated mutations in previously reported retinal dystrophy-causing genes. Methods: A multi-generation caucasian kindred with several members affected by macular dystrophy was identified. After complete ophthalmic examinations of 32 individuals who participated in the study, six were found to be affected with macular degeneration. The disease gene is transmitted as an autosomal dominant trait. Linkage analysis to EFEMP1, RDS/Peripherin, CORD7, STGD3/ELOVL4, ABCA4, dominant drusen on chromosome 6q, and MCDR1 was performed. Results: Affected individuals showed areas of macular geographic atrophy with macular and extramacular drusen. Vision ranged from 20/25- to HM with most affected eyes having visual acuity between 20/200 and 20/400. Linkage analysis using short-tandem polymorphic markers encompassing candidate loci showed that none of above described loci were associated with the phenotype in this kindred. Direct sequencing of RDS/peripherin and ELOVL4 showed no mutations in any of the affected individuals. Conclusion: We identified a kindred with an autosomal dominant macular dystrophy. Linkage analysis and candidate gene sequencing have excluded previously reported genes causing macular dystrophy. A genome-wide scan is underway to identify the potentially novel locus associated with this macular dystrophy.

Keywords: 562 retinal degenerations: hereditary • 335 candidate gene analysis • 418 gene mapping 
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