December 2002
Volume 43, Issue 13
Free
ARVO Annual Meeting Abstract  |   December 2002
Molecular Cloning and Expression of a Phosphatidylinositol Phosphate Kinase From Rat Retina
Author Affiliations & Notes
  • Y-Z Le
    Departments of Cell Biology and Ophthalmology University of Oklahoma Health Sciences Center and Dean McGee Eye Institute Oklahoma City OK
  • M Agbaga
    Departments of Cell Biology and Ophthalmology University of Oklahoma Health Sciences Center and Dean McGee Eye Institute Oklahoma City OK
  • RE Anderson
    Departments of Cell Biology and Ophthalmology University of Oklahoma Health Sciences Center and Dean McGee Eye Institute Oklahoma City OK
  • Footnotes
    Commercial Relationships   Y. Le, None; M. Agbaga, None; R.E. Anderson, None. Grant Identification: NIH NEI (00871, 12190), Research to Prevent Blindness Inc, and Foundation Fighting Blindness
Investigative Ophthalmology & Visual Science December 2002, Vol.43, 1438. doi:
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      Y-Z Le, M Agbaga, RE Anderson; Molecular Cloning and Expression of a Phosphatidylinositol Phosphate Kinase From Rat Retina . Invest. Ophthalmol. Vis. Sci. 2002;43(13):1438.

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Abstract

Abstract: : Purpose: Phosphatidylinositol phosphate kinase (PIPK) is an enzyme responsible for the synthesis of PI-4,5-P<sub≷2</sub≷, an important intermediate in the phosphoinositide second messenger pathway. To study the mechanism of replenishment of PI-4,5-P<sub≷2</sub≷ and the activation of PIPK in rod outer segments, we cloned the PIPK gene that is expressed in rat retina. Methods: The rat PIPK cDNA was cloned using a RT-PCR based approach. The entire open reading frame was sequenced, fused to the glutathione S-transferase (GST) gene, and expressed in <i≷E. coli</i≷. Results: Sequence analysis indicated that the cloned PIPK gene had a high degree of homology with other mammalian PIPKs at the DNA and protein level. The fusion protein was purified with GST-affinity chromatography. Western blot analysis showed that the fusion protein had the similar immunoreactivity to a known Type II PIPK characterized previously in our laboratory. Enzymatic assay of the fusion protein using PI-4-P and PI-5-P as substrates suggested that PI-5-P was the preferable substrate for the GST-PIPK protein. Conclusion: We have cloned and expressed an enzymatically active PIPK from rat retina.

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