December 2002
Volume 43, Issue 13
Free
ARVO Annual Meeting Abstract  |   December 2002
Lipid and DNA Oxidation in Cataracts in Farmed North Atlantic salmon
Author Affiliations & Notes
  • RL Peiffer
    Ophthalmology Small Animal Clinical Sciences
    University of North Carolina Chapel Hill NC
  • E Bjerkas
    Ophthalmology Small Animal Clinical Sciences
    Norwegian School of Veterinary Science Oslo Norway
  • I Bjerkas
    Nutrition Morphology
    Norwegian School of Veterinary Science Oslo Norway
  • CD Albright
    Nutrition Morphology
    University of North Carolina Chapel Hill NC
  • Footnotes
    Commercial Relationships   R.L. Peiffer, None; E. Bjerkas, None; I. Bjerkas, None; C.D. Albright, None.
Investigative Ophthalmology & Visual Science December 2002, Vol.43, 2367. doi:
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    • Get Citation

      RL Peiffer, E Bjerkas, I Bjerkas, CD Albright; Lipid and DNA Oxidation in Cataracts in Farmed North Atlantic salmon . Invest. Ophthalmol. Vis. Sci. 2002;43(13):2367.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose: To investigate the role of oxidative damage in cataractogenesis in the lenses of farm-raised North Atlantic Salmon fed a controlled diet. Methods:Following clinical examination and characterization of cataracts lenses were harvested and fixed routinely for morphologic studies. In addition, immunohistochemistry using markers for hydroxynoneal, a lipid oxidation product, and guanine oxidation product was performed on 6 micron, parrafin-embedded sections. Results:Oxidative damage was evident in the epithelium and in a perinuclear cortical zone which corresponded with clinical and histopathologic alterations of cataract. Conclusion:Cataract in farmed fish is a production-related disease of economic significance and multifactorial origin including nutrition and enviornmental factors such as water temperature. Altered glucose metabolism has been suggested to play a role in this model of cataractogenesis. The above results implicate oxidation of both lipids and nucleic acid as contributatory mechanisms.

Keywords: 338 cataract • 504 oxidation/oxidative or free radical damage 
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