December 2002
Volume 43, Issue 13
Free
ARVO Annual Meeting Abstract  |   December 2002
TIP120A in RPE Cells is Increased by the Addition of Retinoic Acid
Author Affiliations & Notes
  • J Lee
    Cole Eye Institute The Cleveland Clinic Foundation Cleveland OH
  • K Nishiyama
    Cole Eye Institute The Cleveland Clinic Foundation Cleveland OH
  • S Yogosawa
    Faculty of Science Chiba University Chiba Japan
  • T Tamura
    Faculty of Science Chiba University Chiba Japan
  • JG Hollyfield
    Cole Eye Institute The Cleveland Clinic Foundation Cleveland OH
  • Footnotes
    Commercial Relationships   J. Lee, None; K. Nishiyama, None; S. Yogosawa, None; T. Tamura, None; J.G. Hollyfield, None. Grant Identification: Support: NIH, FFB and RRF
Investigative Ophthalmology & Visual Science December 2002, Vol.43, 2424. doi:
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      J Lee, K Nishiyama, S Yogosawa, T Tamura, JG Hollyfield; TIP120A in RPE Cells is Increased by the Addition of Retinoic Acid . Invest. Ophthalmol. Vis. Sci. 2002;43(13):2424.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose: TIP120A is a transcription factor in eukaryotic cells. It is thought to coordinate gene expression during cell growth and/or differentiation. In the visual system, TIP120A has not been studied and its function is not known. This study evaluates that TIP120A in the retina, RPE and RPE/choroid and follows changes in the expression after retinoic acid treatment. Methods: Eyes (adult BALB/c and C57BL/6J mice and adult Sprague-Dawley rats) were fixed in 4% paraformaldehyde and prepared for immunohistochemical analysis. Rat RPE-J cells were incubated for 8 days with/without retinoic acid. Total RNA was isolated and TIP120A mRNA changes were studied by RT-PCR. TIP120A polyclonal antibody was prepared in rabbit. Western blot analysis of the RPE-J cells with/without retinoic acid was performed. Results: TIP120A is strongly immunoreactive in RPE cells. RPE cell nuclei are intensively stained. RT-PCR and Western blot analysis indicated that TIP120A is expressed in RPE-J cells. Furthermore, TIP120A increased following retinoic acid treatment in both mRNA and protein. Immunoprecipitation showed that TIP120A forms a physical complex with TATA-binding protein in RPE-J cells. Conclusion: TIP120A is expressed in RPE cells. Endogenous TIP120A and TATA-binding protein interact in RPE J-cells. The present data suggest that TIP120A may play a regulatory role in RPE cells, possibly through interacting with TATA-binding protein. The prominent TIP120A in the RPE and the interaction with TATA-binding protein suggest that additional studies are needed to clarify the role and function of this regulatory molecule in retinal tissues.

Keywords: 567 retinal pigment epithelium • 605 transcription factors 
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