December 2002
Volume 43, Issue 13
Free
ARVO Annual Meeting Abstract  |   December 2002
Molecular Signatures of Infectious Agents in Ocular Neoplasms
Author Affiliations & Notes
  • RR Buggage
    Laboratory of Immunology
    National Eye Institute/National Institutes of Health Bethesda MD
  • D Shen
    Laboratory of Immunology
    National Eye Institute/National Institutes of Health Bethesda MD
  • N Tuaillon
    Laboratory of Immunology
    National Eye Institute/National Institutes of Health Bethesda MD
  • GA Levy-Clarke
    Laboratory of Immunology
    National Eye Institute/National Institutes of Health Bethesda MD
  • JA Smith
    Division of Epidemiology and Clinical Research
    National Eye Institute/National Institutes of Health Bethesda MD
  • C-C Chan
    Laboratory of Immunology
    National Eye Institute/National Institutes of Health Bethesda MD
  • Footnotes
    Commercial Relationships   R.R. Buggage, None; D. Shen, None; N. Tuaillon, None; G.A. Levy-Clarke, None; J.A. Smith, None; C. Chan, None.
Investigative Ophthalmology & Visual Science December 2002, Vol.43, 2591. doi:
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    • Get Citation

      RR Buggage, D Shen, N Tuaillon, GA Levy-Clarke, JA Smith, C-C Chan; Molecular Signatures of Infectious Agents in Ocular Neoplasms . Invest. Ophthalmol. Vis. Sci. 2002;43(13):2591.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose: Infectious agents are known to cause a wide variety of recognizable ocular infections that can be diagnosed clinically and treated with specific antibiotic therapy. There are several ocular conditions including neoplastic, degenerative and autoimmune diseases in which infectious pathogens have been implicated in the disease pathogenesis. Using molecular analysis we sought to investigate the role of infectious agents in ocular neoplasms. Methods: Ocular tumors from patients known to have or suspected to have a systemic infectious disease were examined with routine histology and immunohistochemistry and diagnosed using standard histopathologic critieria. Microdissection [either by laser capture (Arcturus) or manually] was performed to remove neoplastic cells from the ocular lesions. PCR for specific infectious agents was performed on the microdissected neoplastic cells and the results were confirmed by Southern blot hybridization. The gene-specific PCR primers were as follows: 5'-GGA ACT GCA TCC GTT CAT GAG-3' sense and 5'-TCT TTA AAG CGT TCG TGG TC-3' antisense for T. gondii B1 gene; 5'-TCC GTG TTG TCT ACG TCC AG-3' sense and 5'-AGC CGA AAG GAT TCC ACC AT-3' antisense for HHV-8; 5'-GAC GAG GGG CCA GGT ACA-3' sense and 5'-GCA GCC AAT GCT TCT TGG ACG T-3' antisense for EBV; 5'-CCA TCAC CAG CAG CTA GAT AGC-3' sense and 5'-AGT TGC TGG TAT TCT CGC CTT AAT-3' antisense for the HTLV-1 gag gene and 5'-AAT TCA TTC AAA CAT CTG AC-3' sense and 5'-GGC AGT TGT TTT AAG AAA AG-3' antisense for the HTLV-1 pol gene. Results: Ocular tumors including a conjunctival papilloma, conjunctival lymphoma, and primary intraocular lymphomas of both T and B-cell phenotype were diagnosed. By conventional pathology no evidence of a microorganism was identified in any of the ocular tumors. Using microdissection and PCR we detected HPV type 33 DNA in the conjunctival papilloma. In two conjunctival lymphomas and one case of intraocular lymphoma from HTLV-1 infected patients we identified the presence of HTLV-1 gene sequences in the neoplastic cells. In the studied cases of B-cell primary intraocular lymphoma we found DNA for HHV-8 (6/32), EBV (2/21), and Toxoplasma gondii (2/16). Conclusion: Our findings confirm the oncogenic potential of infectious agents in the eye and raise the possibility that both benign and malignant ocular neoplasms can possibly be prevented or treated with antibiotic therapy.

Keywords: 468 microbial pathogenesis: clinical studies • 610 tumors • 507 pathology: human 
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