December 2002
Volume 43, Issue 13
Free
ARVO Annual Meeting Abstract  |   December 2002
Autosomal dominant Stargardt Macular Dystrophy: clinical features and linkage analysis in a large Greek Family
Author Affiliations & Notes
  • K-K Fan
    Ophthalmic Research Cole Eye Institute Cleveland Clinic Foundation Cleveland OH
  • GE Kenney
    Gilmore Academy Gates Mill OH
  • G Kitsos
    Department of Ophthalmology University of Ioannina Ioannina Greece
  • Z Yang
    Ophthalmic Research Cole Eye Institute Cleveland Clinic Foundation Cleveland OH
  • E Economou-Petersen
    Drakopoulion Blood Bank Center Athens Greece
  • M Grigoriadou
    Department of Genetics Institute of Child Health Athens Greece
  • DJ Zack
    Wilmer Eye Institute Johns Hopkins University Baltimore MD
  • K Psilas
    Department of Ophthalmology University of Ioannina Ioannina Greece
  • MB Petersen
    Department of Genetics Institute of Child Health Athens Greece
  • K Zhang
    Ophthalmic Research Cole Eye Institute Cleveland Clinic Foundation Cleveland OH
  • Footnotes
    Commercial Relationships   K. Fan, None; G.E. Kenney, None; G. Kitsos, None; Z. Yang, None; E. Economou-Petersen, None; M. Grigoriadou, None; D.J. Zack, None; K. Psilas, None; M.B. Petersen, None; K. Zhang, None. Grant Identification: Cole Eye Institute, NIH, Steinbach Fund, and Fight For Sight
Investigative Ophthalmology & Visual Science December 2002, Vol.43, 2828. doi:
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    • Get Citation

      K-K Fan, GE Kenney, G Kitsos, Z Yang, E Economou-Petersen, M Grigoriadou, DJ Zack, K Psilas, MB Petersen, K Zhang; Autosomal dominant Stargardt Macular Dystrophy: clinical features and linkage analysis in a large Greek Family . Invest. Ophthalmol. Vis. Sci. 2002;43(13):2828.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose:Stargardt macular dystrophy (STGD) is the most common hereditary macular dystrophy and is characterized by decreased central vision in the 1st or 2nd decade of life, macular atrophy, and presence of yellow flecks in the posterior pole. In most instances STGD is inherited as an autosomal recessive or simplex trait. A recessive locus was assigned to chromosome 1p (STGD1, ABCA4 gene). Numerous families have been described in which features of STGD are transmitted in an autosomal dominant manner, and dominant loci have been mapped to chromosomes 6q (STGD3, ELOVL4 gene) and 4p (STGD4). We describe the effort to map a disease-causing gene in a large Greek family using genetic linkage analysis. Methods:Ophthalmoscopic examination, fundus photography, and fluorescein angiography were performed to identify individuals with STGD. Genomic DNA was obtained from the blood of 29 family members (11 affected, 18 unaffected) and was genotyped using polymorphic DNA markers. Results:We describe a large five-generation pedigree with dominant STGD from Epirus, Greece. The family consists of 234 individuals with 24 affected members. Clinical examination revealed visual acuity ranging from 20/25 to 20/200, with age at onset between 7 and 18 years. Fundoscopic examination showed loss of foveal reflex, "beaten bronze" appearance of the foveal region, and flecks of varying degrees. The disease was symmetric in the two eyes and progressive with age. Linkage analysis with short tandem repeat (STR) markers excluded linkage to known STGD loci, and a genomewide search is in progress. Conclusion:We have identified a family with an autosomal dominant STGD likely representing a new disease locus. Identification of another causative gene for STGD will aid in our understanding of the pathogenesis of this disease, and allow the investigation of therapeutic agents aimed at treating the underlying biochemical cause of Stargardt’s and other retinal dystrophies.

Keywords: 418 gene mapping • 457 linkage analysis • 420 genetics 
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