December 2002
Volume 43, Issue 13
Free
ARVO Annual Meeting Abstract  |   December 2002
Treatment With Alpha-MSH Reduces Corneal Allorejection
Author Affiliations & Notes
  • P Hamrah
    Schepens Eye Research Institute and Department of Ophthalmology Harvard Medical School Boston MA
  • Z Haskova
    Schepens Eye Research Institute and Department of Ophthalmology Harvard Medical School Boston MA
  • Q Zhang
    Schepens Eye Research Institute and Department of Ophthalmology Harvard Medical School Boston MA
  • AW Taylor
    Schepens Eye Research Institute and Department of Ophthalmology Harvard Medical School Boston MA
  • B Ksander
    Schepens Eye Research Institute and Department of Ophthalmology Harvard Medical School Boston MA
  • MR Dana
    Schepens Eye Research Institute and Department of Ophthalmology Harvard Medical School Boston MA
  • Footnotes
    Commercial Relationships   P. Hamrah, None; Z. Haskova, None; Q. Zhang, None; A.W. Taylor, None; B. Ksander, None; M.R. Dana, None. Grant Identification: Support: NIH Grant EY12963, Research to Prevent Blindness, and Fight for Sight
Investigative Ophthalmology & Visual Science December 2002, Vol.43, 2961. doi:
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    • Get Citation

      P Hamrah, Z Haskova, Q Zhang, AW Taylor, B Ksander, MR Dana; Treatment With Alpha-MSH Reduces Corneal Allorejection . Invest. Ophthalmol. Vis. Sci. 2002;43(13):2961.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose:α-Melanocyte stimulating hormone (α-MSH) is a proopiomelanocortin-derived neuropeptide that suppresses mammalian host inflammatory defense mechanisms. The purpose of this study was to determine the possible role of α-MSH on orthotopic corneal allograft survival and the mechanisms by which it may influence graft outcome. Methods:Orthotopic corneal transplantation was performed using corneal buttons of C56BL/6 mice grafted into BALB/c recipients. Recipients either received a sham injection or were treated with 2.0 µg subconjunctival injections of α-MSH twice weekly. Grafts were followed for 70 days and graft inflammation/opacification was compared between the two groups in a masked fashion. Additionally, graft infiltration was determined in both groups at days 7 and 14. Ocular gene expression of select inflammatory cytokines was evaluated at different timepoints by the ribonuclease protection assay. Additionally, allospecific delayed type hypersensitivity (DTH) was compared among the groups 3 weeks post transplantation. Results:Corneal allografts in α-MSH-treated recipients showed an increase in graft survival as compared to untreated hosts. Over 70% of allografts in α-MSH treated hosts survived at 70 days, compared to less than 50% in the untreated group. Graft infiltration by mononuclear and polymorphonuclear (PMN) cells was significantly decreased in α-MSH treated mice compared to untreated hosts (P<0.05). Moreover, DTH results and cytokine expression (including Interferon-g and Interleukin-2) were significantly reduced in α-MSH treated mice as compared to untreated recipients. Conclusion:We provide, for the first time, in vivo evidence that treatment with α-MSH may significantly reduce allorejection of corneal transplants.

Keywords: 607 transplantation • 369 cornea: clinical science • 488 neuropeptides 
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