July 2019
Volume 60, Issue 9
Open Access
ARVO Annual Meeting Abstract  |   July 2019
A whole exome sequencing-based panel assay with boosted clinical content generates a high diagnostic yield in patients with inherited eye diseases
Author Affiliations & Notes
  • Kati Kämpjärvi
    Blueprint Genetics, Helsinki, Finland
  • Kirsty Wells
    Blueprint Genetics, Helsinki, Finland
  • Miika Mehine
    Blueprint Genetics, Helsinki, Finland
  • Johanna Känsäkoski
    Blueprint Genetics, Helsinki, Finland
  • Laura Sarantaus
    Blueprint Genetics, Helsinki, Finland
  • Hanna Västinsalo
    Blueprint Genetics, Helsinki, Finland
  • Jennifer Schleit
    Blueprint Genetics, San Francisco, California, United States
  • Inka Saarinen
    Blueprint Genetics, Helsinki, Finland
  • Mikko Muona
    Blueprint Genetics, Helsinki, Finland
  • Samuel Myllykangas
    Blueprint Genetics, Helsinki, Finland
  • Tero-Pekka Alastalo
    Blueprint Genetics, San Francisco, California, United States
  • Juha Koskenvuo
    Blueprint Genetics, Helsinki, Finland
  • Jussi Paananen
    Blueprint Genetics, Helsinki, Finland
  • Sari Tuupanen
    Blueprint Genetics, Helsinki, Finland
  • Footnotes
    Commercial Relationships   Kati Kämpjärvi, Blueprint Genetics (E); Kirsty Wells, Blueprint Genetics (E); Miika Mehine, Blueprint Genetics (E); Johanna Känsäkoski, Blueprint Genetics (E); Laura Sarantaus, Blueprint Genetics (E); Hanna Västinsalo, Blueprint Genetics (E); Jennifer Schleit, Blueprint Genetics (E); Inka Saarinen, Blueprint Genetics (E); Mikko Muona, Blueprint Genetics (E); Samuel Myllykangas, Blueprint Genetics (S); Tero-Pekka Alastalo, Blueprint Genetics (S); Juha Koskenvuo, Blueprint Genetics (S); Jussi Paananen, Blueprint Genetics (E); Sari Tuupanen, Blueprint Genetics (E)
  • Footnotes
    Support  None
Investigative Ophthalmology & Visual Science July 2019, Vol.60, 421. doi:
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      Kati Kämpjärvi, Kirsty Wells, Miika Mehine, Johanna Känsäkoski, Laura Sarantaus, Hanna Västinsalo, Jennifer Schleit, Inka Saarinen, Mikko Muona, Samuel Myllykangas, Tero-Pekka Alastalo, Juha Koskenvuo, Jussi Paananen, Sari Tuupanen; A whole exome sequencing-based panel assay with boosted clinical content generates a high diagnostic yield in patients with inherited eye diseases. Invest. Ophthalmol. Vis. Sci. 2019;60(9):421.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Genetic testing is essential in the diagnosis of inherited eye diseases (IEDs), due to their genetic heterogeneity and phenotypic overlap, and the need to establish a molecular diagnosis for targeted therapy selection. However, IED diagnostics can be compromised by tests which are poorly validated or do not capture the full spectrum of causative genetic variation. We aimed to develop and validate a high quality whole exome sequencing (WES) based platform, with boosted clinical content tailored to IEDs, and to assess diagnostic yield in a large IED cohort.

Methods : Performance of an xGEN Exome Research Panel assay, with bespoke clinical content, was tested through next generation sequencing (NGS) of reference samples on the Illumina NovaSeq platform. The assay was designed to cover clinically-relevant intronic and difficult-to-sequence regions, and detect copy number variants (CNVs). Based on this assay, 23 ophthalmology gene panels were curated, covering 438 unique genes, and their diagnostic performance evaluated.

Results : In clinically-relevant genes, the assay showed high average sequencing depth (174x) and coverage (99.4% regions covered >20x). Analytic validation showed excellent sensitivity for SNVs (0.997), INDELs 2-50 base pairs (0.988), 1-exon CNVs (0.92), and 5-exon CNVs (0.99). The assay showed uniform coverage over difficult-to-sequence regions, including RPGR ORF15 and GC-rich 1st exons. The Usher Syndrome Panel gave the highest diagnostic yield (>80%) and the Glaucoma Panel the lowest (11%). A 266-gene Retinal Dystrophy Panel, the largest and most frequently ordered panel, yielded a diagnosis in 58% of (1587) cases. Diagnoses were made in 111 genes in total, the most frequently implicated genes being ABCA4, USH2A, RPGR, RHO, and PRPH2. Diagnoses were also made in genes newly implicated in IEDs such as CTNN1A, TTLL5, and RAB28. Approximately 3% of cases had a diagnostic CNV. Disease-causing variants were detected in RPGR ORF15 and in custom targeted deep intronic regions.

Conclusions : A WES assay with boosted clinical content provides high sequencing coverage, depth, sensitivity and specificity, and provides high diagnostic yields for IED patients. The genetic variability of diagnoses in our cohort supports the use of comprehensive NGS panels in IED diagnostics, to optimize diagnostic yield and clinical care.

This abstract was presented at the 2019 ARVO Annual Meeting, held in Vancouver, Canada, April 28 - May 2, 2019.

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