July 2019
Volume 60, Issue 9
Open Access
ARVO Annual Meeting Abstract  |   July 2019
High myopia and strabismus induced by a deep intronic mutation in COL2A1
Author Affiliations & Notes
  • Shirel Weiss
    the Krieger eye research laboratory , Tel Aviv University , Petach Tiqwa, Israel
    Rapport faculty of medicine, Technion, Haifa, Israel
  • Naama Orenstein
    Genetics, Schneider Children's medical center, Petach Tiqwa, Israel
  • Alon Zahavi
    Ophthalmology, Rabin Medical Center, Petach Tiqwa, Israel
  • Nitza Goldenberg-Cohen
    the Krieger eye research laboratory , Tel Aviv University , Petach Tiqwa, Israel
    Rapport faculty of medicine, Technion, Haifa, Israel
  • Footnotes
    Commercial Relationships   Shirel Weiss, None; Naama Orenstein, None; Alon Zahavi, None; Nitza Goldenberg-Cohen, None
  • Footnotes
    Support  Israel affiliated Arvo chapter
Investigative Ophthalmology & Visual Science July 2019, Vol.60, 425. doi:
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    • Get Citation

      Shirel Weiss, Naama Orenstein, Alon Zahavi, Nitza Goldenberg-Cohen; High myopia and strabismus induced by a deep intronic mutation in COL2A1. Invest. Ophthalmol. Vis. Sci. 2019;60(9):425.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Stickler syndrome is an autosomal dominant connective tissue disorder with systemic and ocular manifestations. Ophthalmological findings include moderate to high myopia, abnormal vitreous, glaucoma and cataract. Three subtypes have been described, distinguished by clinical characteristics and specific genetic mutations.
We describe a possible genetic mutation leading to high myopia, strabismus and retinal detachment in multiple family members, compatible with Stickler syndrome.

Methods : Clinical and genetic analysis was done for a family suspected of Stickler syndrome. A detailed pedigree, clinical examination, blood sampling and DNA extraction, whole exome sequencing, and a bioinformatics analysis were performed.

Results : Demographic and clinical data of 5 generations of the family were collected. Specific clinical phenotype of Stickler syndrome documented in 13 family members, with 7 having more than one clinical feature. A genetic analysis was performed for 4 family members – proband (the patient), mother, maternal grandfather, and healthy father. As whole exome analysis was insufficient in detecting a relevant mutation, anlysis was extended to include intronic areas. A deep intronic mutation COL2A1 c.1527+135G>A was discovered.

Conclusions : Stickler syndrome has several subtypes with variable clinical features, thereby making a prediction on disease gene locus based on clinical characteristics challenging. Here we present a rarely described intronic mutation in the COL2A1 gene. An early diagnosis of the condition may be aided by genetic testing and is important for genetic counseling, proper clinical management and improved prognosis.

This abstract was presented at the 2019 ARVO Annual Meeting, held in Vancouver, Canada, April 28 - May 2, 2019.

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