July 2019
Volume 60, Issue 9
Open Access
ARVO Annual Meeting Abstract  |   July 2019
Targeting the leukotriene B4 pathway and/or complement C5 via dual-functional recombinant rVA576 (Coversin) in Experimental Autoimmune Uveitis (EAU)
Author Affiliations & Notes
  • Mali Eskandarpour
    Ocular Immunology, UCL Institute of Ophthalmology, London, United Kingdom
  • Xiaozhe Zhang
    Ocular Immunology, UCL Institute of Ophthalmology, London, United Kingdom
  • Grazyna Galatowics
    Ocular Immunology, UCL Institute of Ophthalmology, London, United Kingdom
  • Miles Nunn
    Akari Therapeutics plc, London, United Kingdom
  • Wynne Weston-Davies
    Akari Therapeutics plc, London, United Kingdom
  • Virginia L Calder
    Ocular Immunology, UCL Institute of Ophthalmology, London, United Kingdom
  • Footnotes
    Commercial Relationships   Mali Eskandarpour, None; Xiaozhe Zhang, None; Grazyna Galatowics, None; Miles Nunn, Akari Therapeutics plc (E); Wynne Weston-Davies, Akari Therapeutics plc (E); Virginia Calder, Akari Therapeutics plc (R), Akari Therapeutics plc (F)
  • Footnotes
    Support  None
Investigative Ophthalmology & Visual Science July 2019, Vol.60, 797. doi:
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      Mali Eskandarpour, Xiaozhe Zhang, Grazyna Galatowics, Miles Nunn, Wynne Weston-Davies, Virginia L Calder; Targeting the leukotriene B4 pathway and/or complement C5 via dual-functional recombinant rVA576 (Coversin) in Experimental Autoimmune Uveitis (EAU). Invest. Ophthalmol. Vis. Sci. 2019;60(9):797.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : To explore the impact of rVA576, a recombinant bifunctional protein that inhibits LTB4 and C5, when administered intravitreally or topically during active EAU.

Methods : To induce EAU, C57B6 mice (female, 5-8 weeks old) were immunized with IRBP1-20 as previously described1. Mice were clinically scored by retinal fundoscopy and, on day 14-16 post immunization, rVA576 (0.5%) or saline was administered intravitreally. Disease progression was graded clinically by retinal fundoscopy and scored histologically. We also investigated the effect of rVA576 on CD4+ T cell subsets using retinal flow cytometry (Fortessa, BD).

Results : We found that rVA576 mitigated disease progression at one day post intravitreal administration as determined by clinical scoring (mean score±SD; 5.167±1.327; n=6) relative to saline controls (10.33 ± 1.202; n=3; P= 0.043). A significant decrease in retinal Th17 cells in the rVA576 treated group (mean percentage±SD; 46.28±7.079; n=6) was detected relative to controls (83.33±4.842; n=3; P=0.015). A decrease in retinal double-positive Th1+Th17+ (RORγt+T-bet+) levels (40.68± 7.505; n=6) was also observed relative to controls (82.40 ± 7.969; n=3; P=0.011). In addition, decreases in RORγt+IFNγ+ cells (28.78±4.527; n=6) were detected in rVA576-treated mice in comparison with controls (51.63 ± 6.583; n=3; P=0.025) after one day post administration while there was no significant difference in the expression levels of CD4+CD25+FoxP3+ and CD4+T-bet+IFNγ+.

Conclusions : rVA576 attenuated EAU progression which was accompanied with a decrease in Th17 and double-positive Th1+Th17+ cells. We plan to test a long-acting version of rVA576, which may offer more prolonged efficacy in murine EAU.
1 Eskandarpour M., et al., (2017)

This abstract was presented at the 2019 ARVO Annual Meeting, held in Vancouver, Canada, April 28 - May 2, 2019.

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