July 2019
Volume 60, Issue 9
Open Access
ARVO Annual Meeting Abstract  |   July 2019
Anatomical response to anti-vascular endothelial growth factor (anti-VEGF) in patients with diabetic macular edema (DME) and its potential “in vitro” prediction by using Induced Pluripotent Stem Cell (IPSC)-derived endothelial cells (ECs).
Author Affiliations & Notes
  • Noemi Lois
    Wellcome-Wolfson Institute for Experimental Medicine, Belfast, Northern Ireland, United Kingdom
    Ophthalmology, Belfast Health and Social Care Trust, Belfast, Northern Ireland, United Kingdom
  • Andriana Margariti
    Wellcome-Wolfson Institute for Experimental Medicine, Belfast, Northern Ireland, United Kingdom
  • Marta Vila-Gonzalez
    Wellcome-Wolfson Institute for Experimental Medicine, Belfast, Northern Ireland, United Kingdom
  • Stephen Stewart
    Ophthalmology, Belfast Health and Social Care Trust, Belfast, Northern Ireland, United Kingdom
  • Gianni Virgili
    Ophthalmology, University of Florence, Florence, Italy
  • Alan W Stitt
    Wellcome-Wolfson Institute for Experimental Medicine, Belfast, Northern Ireland, United Kingdom
  • Footnotes
    Commercial Relationships   Noemi Lois, None; Andriana Margariti, None; Marta Vila-Gonzalez, None; Stephen Stewart, None; Gianni Virgili, None; Alan Stitt, None
  • Footnotes
    Support  Medical Research Council, UK
Investigative Ophthalmology & Visual Science July 2019, Vol.60, 3864. doi:
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      Noemi Lois, Andriana Margariti, Marta Vila-Gonzalez, Stephen Stewart, Gianni Virgili, Alan W Stitt; Anatomical response to anti-vascular endothelial growth factor (anti-VEGF) in patients with diabetic macular edema (DME) and its potential “in vitro” prediction by using Induced Pluripotent Stem Cell (IPSC)-derived endothelial cells (ECs).. Invest. Ophthalmol. Vis. Sci. 2019;60(9):3864.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : To phenotype DME based on anatomical response to anti-VEGF and evaluate the predictive value of in vitro readouts of iPSC-ECs.

Methods : Medical records and serial optical coherence tomography scans of consecutive patients with DME initiating treatment with Ranibizumab at the Belfast Trust between March and December 2014 were reviewed. Patients were eligible if they had received a loading dose (three injections) and were followed for at least 12 months. Based on the treatment response patients were classified as “responders” (retina fully dry at any time during the course of treatment), “partial responders” (fluid decreased but never cleared) or “non-responders” (fluid unchanged). Differences in clinical characteristics among the three groups were statistically sought. iPSCs were generated from blood cells obtained from three "responders" and three "non-responders" and differentiated into ECs. Phosphorylation of the VEGF receptor (VEGFR2) and gene expression profiles were evaluated in these groups.

Results : There were 100 DME patients (140 eyes), classified as “responders” (45 eyes, 32%), “partial responders” (86 eyes, 61%) or “non-responders" (9 eyes, 6%) with no differences in clinical characteristics among them. When stimulated with VEGF, “non-responders” derived iPSC-ECs showed increased VEGFR2 phosphorylation when compared with “responders”. Furthermore, gene expression profiles of iPSC-ECs from “responders” and “non-responders” demonstrated a number of differentially expressed gene transcripts that were linked with cell permeability and VEGF signalling.

Conclusions : In only ~ a third of patients DME fully cleared after a minimum of one year of treatment suggesting that other mechanisms besides VEGF-induced leakage are involved in DME in many patients. Patient-specific iPSC-EC responses in vitro differred between "responders" and "non-responders". This “patient-in-a dish” approach has potential to be used to predict clinical responses to anti-VEGF therapy in DME.

This abstract was presented at the 2019 ARVO Annual Meeting, held in Vancouver, Canada, April 28 - May 2, 2019.

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