July 2019
Volume 60, Issue 9
Open Access
ARVO Annual Meeting Abstract  |   July 2019
Selective retinal ganglion cell vulnerability in older mice exposed to acute intraocular pressure elevation and the potential involvement of the P2X7-receptor
Author Affiliations & Notes
  • Anna Yaomei Wang
    Department of Anatomy and Neuroscience, University of Melbourne, Melbourne, Victoria, Australia
  • Kirstan Anne Vessey
    Department of Anatomy and Neuroscience, University of Melbourne, Melbourne, Victoria, Australia
  • Bang V Bui
    Department of Optometry and Vision Sciences, University of Melbourne, Melbourne, Victoria, Australia
  • Vickie Hoi Ying Wong
    Department of Optometry and Vision Sciences, University of Melbourne, Melbourne, Victoria, Australia
  • Pei Ying Lee
    Department of Optometry and Vision Sciences, University of Melbourne, Melbourne, Victoria, Australia
  • Erica L Fletcher
    Department of Anatomy and Neuroscience, University of Melbourne, Melbourne, Victoria, Australia
  • Footnotes
    Commercial Relationships   Anna Wang, None; Kirstan Vessey, None; Bang Bui, None; Vickie Wong, None; Pei Ying Lee, None; Erica Fletcher, None
  • Footnotes
    Support  National Health and Medical Research Council (NHMRC) grant: APP 1138509
Investigative Ophthalmology & Visual Science July 2019, Vol.60, 4846. doi:
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      Anna Yaomei Wang, Kirstan Anne Vessey, Bang V Bui, Vickie Hoi Ying Wong, Pei Ying Lee, Erica L Fletcher; Selective retinal ganglion cell vulnerability in older mice exposed to acute intraocular pressure elevation and the potential involvement of the P2X7-receptor. Invest. Ophthalmol. Vis. Sci. 2019;60(9):4846.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : P2X7-receptors may contribute to retinal ganglion cell (RGC) death in glaucoma. We examined RGC function following acute intraocular pressure (IOP) elevation in older C57BL/6 (WT) mice and P2X7-receptor knockout (P2X7-KO) mice using a multielectrode array (MEA).

Methods : In 13-month-old WT (n=15) and P2X7-KO mice (n=9), the anterior chamber of one eye was cannulated (50μm glass micropipette connected to a height-adjustable Hanks balanced salt solution reservoir) to increase IOP to 50 mmHg for 30 minutes. The contralateral eye was cannulated without increasing IOP (sham). Three days following injury, mice were dark-adapted over-night and retinae were mounted onto an MEA to record RGC spontaneous activityand light-evoked responses. Full field stimuli were 1 second flashes modulated between 0 and 1066 photoisomerisations/rod/sec. To test frequency responsiveness, full field light ON and OFF components were modulated from 1 to 30 Hz. Receptive fields were mapped by calculating the spike triggered average in response to a 32x32 checkerboard stimulus (70µm squares) presented at 12 Hz, with mean luminance of 517 photoisomerations/rod/sec. Cells were analyzed and sorted using Spike2 and classified into ON, OFF, ON-OFF and non-responsive types based on peak firing during light on and off full-field stimuli.

Results : In WT mice there was a significant reduction in spontaneous activity (p<0.05) and full-field-evoked spike rates (p<0.05) for OFF RGCs after IOP stress compared to OFF cells of sham eyes. These changes appear to be subtype-specific as ON and ON-OFF cells showed no change in response. There were no further effects of IOP at higher temporal frequencies of full field stimulus, nor were there changes in receptive field size. In P2X7-KO mice, OFF RGCs in IOP stressed eyes showed significantly reduced spontaneous rate (p<0.05) compared to OFF RGCs in WT sham eyes, much like the effect of IOP stress on WT OFF cells. Additionally, ON RGCs from P2X7-KO eyes subjected to IOP stress showed a significant decrease in peak spike rate compared to P2X7-KO sham eyes (p<0.05).

Conclusions : These results suggest that even a short period of mild IOP stress can have long lasting effect on RGC function, particularly that of OFF-RGCs. In contrast to previous studies, P2X7-KO did not prevent RGC functional deficits associated with acute mild IOP elevation.

This abstract was presented at the 2019 ARVO Annual Meeting, held in Vancouver, Canada, April 28 - May 2, 2019.

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