July 2019
Volume 60, Issue 9
Open Access
ARVO Annual Meeting Abstract  |   July 2019
A PLGA-Carboxyamidotriazole (CAI) Polymeric Suspension Suppresses Pathological Ocular Angiogenesis in rodent CNV model.
Author Affiliations & Notes
  • Sergio Li Calzi
    Ophthalmology and Visual Science, University of Alabama at Birmingham, Birmingham, Alabama, United States
  • DIBYENDU CHAKRABORTY
    Ophthalmology and Visual Science, University of Alabama at Birmingham, Birmingham, Alabama, United States
  • Bright Asare-Bediako
    Ophthalmology and Visual Science, University of Alabama at Birmingham, Birmingham, Alabama, United States
  • Isabel Franklin
    Auburn University, Auburn, Alabama, United States
  • Srikanth Kakumanu
    None, Alabama, United States
  • Laurent Balenci
    None, Ontario, Canada
  • Maria B Grant
    Ophthalmology and Visual Science, University of Alabama at Birmingham, Birmingham, Alabama, United States
  • Alan Franklin
    None, Alabama, United States
  • Footnotes
    Commercial Relationships   Sergio Li Calzi, Alcon (C); DIBYENDU CHAKRABORTY, Alcon (C); Bright Asare-Bediako, Alcon (C); Isabel Franklin, Alcon (C); Srikanth Kakumanu, None; Laurent Balenci, RFE Pharma (E); Maria Grant, Alcon (C); Alan Franklin, Alcon (C), RFE Pharma (E)
  • Footnotes
    Support  none
Investigative Ophthalmology & Visual Science July 2019, Vol.60, 5411. doi:
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      Sergio Li Calzi, DIBYENDU CHAKRABORTY, Bright Asare-Bediako, Isabel Franklin, Srikanth Kakumanu, Laurent Balenci, Maria B Grant, Alan Franklin; A PLGA-Carboxyamidotriazole (CAI) Polymeric Suspension Suppresses Pathological Ocular Angiogenesis in rodent CNV model.. Invest. Ophthalmol. Vis. Sci. 2019;60(9):5411.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : CAI has been used as a systemic anti-angiogenic agent in 900+ patients with different systemic cancers. The purpose of this study was to formulate a potential sustained release safe anti-angiogenic molecule.

Methods : A polymeric Poly lactic co-glycolic acid(PLGA)-Carboxyamidotriazole(CAI) suspension was engineered by double emulsion. To induce choroidal neovascularization, laser injury was made utilizing an Iridex laser at settings of 50µm spot, 100msec duration, and 200 mW power. The resulting choroidal neovascular membrane volume was measured by a masked examiner using bioptigen InVivoVue 2.4.34 software.

Results : Injection of the polymeric PLGA-CAI suspension into the rabbit vitreous caused no toxicity based on retinal exam and histopathology. No inflammation or change in intraocular pressure were noted after injection. No abnormal blood chemistry or internal organ abnormalities were detected following intravitreal administration. At both 1 and 2 weeks after laser injury in mice, intravitreal injection of the PLGA-CAI polymer at a dose range from 500nM-10 µM significantly decreased choroidal neovascular volume in a dose dependent fashion. [mg1] We were able to achieve a 70% reduction in choroidal neovascular volume with this dose range. There was not a significant increase of anti-angiogenic activity observed when this dose range of PLGA-CAI was combined with intravitreal injection of 2µl of aflibercept.

Conclusions : A stable synthetic anti-angiogenic molecule, CAI, has been formulated into a polymeric PLGA suspension. Based on manipulation of particle size and polymer composition, we speculate that PLGA-CAI can be designed to deliver either induction or sustained release therapy, thus addressing a significant unmet clinical need by reducing treatment burden of anti-angiogenic therapy.

This abstract was presented at the 2019 ARVO Annual Meeting, held in Vancouver, Canada, April 28 - May 2, 2019.

 

Intravitreal injection of CAI-PLGA Polymer decreases choroidal neovascular size in a dose dependent fashion. Significant inhibition, p < 0.05, was observed at both doses and time points except for the 2 week post injection timepoint for CAI 500nM. No significant inhibition was noted 1 week after Eylea® injection, but at 2 weeks post injection, an approximate 50% reduction was observed. Overall, the reduction observed by CAI was greater, 65% at 1 week, and 70% at 2 weeks. However, no additional reduction in choroidal neovascular volume was observed when both agents were injected.

Intravitreal injection of CAI-PLGA Polymer decreases choroidal neovascular size in a dose dependent fashion. Significant inhibition, p < 0.05, was observed at both doses and time points except for the 2 week post injection timepoint for CAI 500nM. No significant inhibition was noted 1 week after Eylea® injection, but at 2 weeks post injection, an approximate 50% reduction was observed. Overall, the reduction observed by CAI was greater, 65% at 1 week, and 70% at 2 weeks. However, no additional reduction in choroidal neovascular volume was observed when both agents were injected.

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