June 2020
Volume 61, Issue 7
Free
ARVO Annual Meeting Abstract  |   June 2020
VITAMIN D3 AND MESO ZEAXANTHIN PROTECT RETINAL PIGMENTED EPITHELIAL CELLS FROM β-AMYLOID DAMAGE
Author Affiliations & Notes
  • Claudio Bucolo
    Biomedical and Biotechnological Sciences, University of Catania, Catania, Italy
  • Francesca Lazzara
    Biomedical and Biotechnological Sciences, University of Catania, Catania, Italy
  • Federica Conti
    Biomedical and Biotechnological Sciences, University of Catania, Catania, Italy
  • Chiara B M Platania
    Biomedical and Biotechnological Sciences, University of Catania, Catania, Italy
  • Filippo Drago
    Biomedical and Biotechnological Sciences, University of Catania, Catania, Italy
  • Footnotes
    Commercial Relationships   Claudio Bucolo, None; Francesca Lazzara, None; Federica Conti, None; Chiara B M Platania, None; Filippo Drago, None
  • Footnotes
    Support  Catania University Piano Triennale Ricerca Linea Intervento2
Investigative Ophthalmology & Visual Science June 2020, Vol.61, 4928. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      Claudio Bucolo, Francesca Lazzara, Federica Conti, Chiara B M Platania, Filippo Drago; VITAMIN D3 AND MESO ZEAXANTHIN PROTECT RETINAL PIGMENTED EPITHELIAL CELLS FROM β-AMYLOID DAMAGE. Invest. Ophthalmol. Vis. Sci. 2020;61(7):4928.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose : To assess effects of Vitamin D3 and meso-zeaxanthin, two molecules used in clinical practice in patients with AMD, in an in-vitro model of AMD. Amyloid-β (Aβ) retinal deposition has been reported in patients with age-related macular degeneration (AMD), and it might play a detrimental role in the development of the disease. Fibrils of Aβ have been identified among the constituents of drusen, which cause oxidative stress and retinal pigmented epithelium (RPE) damage.

Methods : Human ARPE-19 cells were treated with soluble Aβ1-42 oligomers; cells were exposed to 1 and 10 μM of Aβ1-42 for 48 h and with vitamin D3 (50 nM-1 μM), meso-zeaxanthin (0.1μM and 0.5 μM) or in combination of the two compounds (Aβ 1 μM-10 μM + vitamin D3 50 nM + meso-zeaxanthin 0.1μM; Aβ 1 μM-10 μM + vitamin D3 1 μM + meso-zeaxanthin 0.5 μM). Cells were pre-treated for 16 hours with both vitamin D3 and meso-zeaxanthin; after pre-treatment ARPE-19 were exposed to Aβ insult. Effects of Aβ and anti-oxidants on ARPE-19 were evaluated with MTT assay and LDH release assay and production of reactive oxygen species (ROS).

Results : Vitamin D3 and meso-zeaxanthin alone or in combination protected ARPE-19 against the damage elicited by amyloid-β (1-10 μM). Aβ significantly (p<0.05) reduced cell proliferation and increased LDH release after 48 h of exposure. These effects were significantly (p<0.05) counteracted by vitamin D3 and meso-zeaxanthin treatments, alone or in combination (the combo treatment exerted the best effect). Further, the release of ROS was significantly (p<0.05) reduced only by vitamin D3 and meso-zeaxanthin co-treatment, in ARPE-19 challenged with Aβ (1-10 μM).

Conclusions : These findings demonstrated, at least in our in-vitro paradigm, that combination of mezo-zeaxanthin/vitamin D3 exerts an higher protective effect compared to the effect of the single molecules in ARPE-19 cells damaged by Aβ. Further studies are needed to confirm the useful combination of meso-zeaxanthin/vitamin D3 to protect RPE cells in different paradigms.

This is a 2020 ARVO Annual Meeting abstract.

×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×