June 2021
Volume 62, Issue 8
Open Access
ARVO Annual Meeting Abstract  |   June 2021
Dissociation enzyme treatment alters strain response of astrocytic lamina in explanted mouse optic nerve head
Author Affiliations & Notes
  • Arina Korneva
    Johns Hopkins Medicine Wilmer Eye Institute, Baltimore, Maryland, United States
  • Elizabeth Cone-Kimball
    Johns Hopkins Medicine Wilmer Eye Institute, Baltimore, Maryland, United States
  • Thao D. Nguyen
    Mechanical Engineering, Johns Hopkins University Whiting School of Engineering, Baltimore, Maryland, United States
    Ophthalmology, Johns Hopkins University School of Medicine, Baltimore, Maryland, United States
  • Harry A Quigley
    Johns Hopkins Medicine Wilmer Eye Institute, Baltimore, Maryland, United States
  • Footnotes
    Commercial Relationships   Arina Korneva, None; Elizabeth Cone-Kimball, None; Thao Nguyen, None; Harry Quigley, None
  • Footnotes
    Support  NIH EY002120, NIH EY001765
Investigative Ophthalmology & Visual Science June 2021, Vol.62, 2375. doi:
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      Arina Korneva, Elizabeth Cone-Kimball, Thao D. Nguyen, Harry A Quigley; Dissociation enzyme treatment alters strain response of astrocytic lamina in explanted mouse optic nerve head. Invest. Ophthalmol. Vis. Sci. 2021;62(8):2375.

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Abstract

Purpose : To measure the effect of short-term exposure to dissociation enzyme on the mechanical response of the optic nerve head (ONH) in explanted mouse eyes.

Methods : The ONH was imaged with two-photon laser scanning microscopy (Zeiss LSM 710) in explanted mouse eyes using previously-published inflation testing methods (FVB/N-Tg(GFAP-GFP)14Mes mice, both sexes, 3-8 months of age). Inflation tests were performed before and again after 60 minutes exposure to TrypLE Express (Gibco 12604013), a non-animal-derived recombinant enzyme and a purified trypsin alternative. These (n=8) were compared to sequential inflation of explants exposed only to buffer (n=8). Deformation of the astrocytic lamina (AL) and of connective tissues of the peripapillary sclera (PPS) was analyzed by digital volume correlation (Korneva et al., 2020). After inflation-testing, treated specimens were prepared either for transmission electron microscopy or for fluorescent immunolabeling with phalloidin to quantify changes in the actin network (Ling et al., 2020).

Results : AL nasal-temporal strain (Exx) increased significantly post TrypLE treatment (post: 3.59±1.10%, pre: 2.52±0.73%, p=0.01). AL superior-inferior strain (Eyy) also significantly increased post TrypLE treatment (post: 2.50±0.64%, pre: 0.86±0.75%, p=002). The change in AL strain after treatment with buffer was insignificant (Exx post: 2.77±1.17%, pre: 2.92±1.41%, p=0.64). The effect of TrypLE on PPS strain response was insignificant (Exx post: 0.00±0.34%, pre: 0.06±0.41%, p=0.93). The differences in PPS strain after TrypLE and buffer were similar (ΔExx p=0.99, ΔEyy p=0.81). The PPS maximum principal strain after TrypLe significantly increased from before treatment (p=0.01) but not compared to after buffer (p=0.67). Quantitative morphological analysis of astrocyte processes in these eyes will be presented.

Conclusions : TrypLe treatment led to greater strain of the AL but similar strain of the PPS in inflation testing of explanted mouse eyes. Morphological studies suggest that astrocyte junctional complexes to the PPS are disrupted in experimental mouse glaucoma eyes (Quillen et al., 2020). Comparisons of experimental alterations of mouse model tissue may elucidate the underlying mechanisms of optic nerve head remodeling.

This is a 2021 ARVO Annual Meeting abstract.

 

Strain map pre (A)and post (B) 60min TrypLE treatment. C Difference in strain (post minus pre) was greater with TrypLE compared to buffer. *p<0.05

Strain map pre (A)and post (B) 60min TrypLE treatment. C Difference in strain (post minus pre) was greater with TrypLE compared to buffer. *p<0.05

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