June 2021
Volume 62, Issue 8
Open Access
ARVO Annual Meeting Abstract  |   June 2021
Role of Plasmacytoid Dendritic Cells in the Homeostasis of Retinal Blood Flow
Author Affiliations & Notes
  • Gustavo Ortiz
    Ophthalmology, Tufts Medical Center, Boston, Massachusetts, United States
  • Arsia Jamali
    Ophthalmology, Tufts Medical Center, Boston, Massachusetts, United States
  • Deshea L Harris
    Ophthalmology, Tufts Medical Center, Boston, Massachusetts, United States
  • Pedram Hamrah
    Ophthalmology, Tufts Medical Center, Boston, Massachusetts, United States
  • Footnotes
    Commercial Relationships   Gustavo Ortiz, None; Arsia Jamali, None; Deshea Harris, None; Pedram Hamrah, None
  • Footnotes
    Support  R01 EY026963, R01EY022695, Massachusetts Lions Eye Research Fund Inc., Tufts Medical Center Institutional Support.
Investigative Ophthalmology & Visual Science June 2021, Vol.62, 652. doi:
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    • Get Citation

      Gustavo Ortiz, Arsia Jamali, Deshea L Harris, Pedram Hamrah; Role of Plasmacytoid Dendritic Cells in the Homeostasis of Retinal Blood Flow. Invest. Ophthalmol. Vis. Sci. 2021;62(8):652.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : We have recently demonstrated the presence of plasmacytoid dendritic cells (pDCs) in murine retina. In particular we have shown that pDCs are in close proximity to retinal vessels. Thus, we aimed to evaluate whether resident pDCs may mediate the homeostasis of retinal vasculature in the laser-induced choroidal neovascularization (CNV) mice model of age-related macular degeneration.

Methods : Six- to 8-week-old, BDCA2-DTR mice were intraperitoneally injected with 200 mg/ml of diphtheria toxin or PBS as pDC-depleted and sham-depleted animals respectively. pDC-depletion was corroborated by flow cytometry. Both naïve and laser-induced CNV depleted animals were evaluated in this study. CNV was performed on the same day of depletion. Three and 7 days after CNV induction, fluorescein angiography was conducted in sham and pDC-depleted animals. Fundus images were obtained; abnormal vascular leakage and scar size were quantified as fluorescence intensity and scar area (as normalized to optic disc) respectively. Further, we used our transgenic DPE-GFP×RAG-1-/- mice in which only pDCs express GFP, to quantify retinal GFP cells in naïve and after laser-induced CNV.

Results : Fluorescein angiography in naïve animals showed that vascular leakage was 1.5-fold (p<0.001) and 1.4-fold (p=0.042) increased after 3 and 7 days of pDC-depletion, respectively, compared with sham depleted. At 7 days after CNV induction, vascular leakage and scar area increased 4-fold (p=0.040) and 2-fold (p=0.046), respectively, in pDC-depleted animals. Additionally, retinal eGFP cells were CD11clow, PDCA-1+, Ly6C+, Ly49Q+ and CD19, CD3, F4/80, CD11b. This population was increased 4-fold after laser-induced CNV as measured by flow cytometry and epi-fluorescent microscopy (p<0.001).

Conclusions : pDCs are required to preserve vasculature homeostasis in naïve retinas. Moreover, pDC depletion in a clinically relevant model of AMD exhibits worse outcomes, including increased vascular leakage and scar size, indicating that pDCs may be necessary to limit disease.

This is a 2021 ARVO Annual Meeting abstract.

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