June 2021
Volume 62, Issue 8
Open Access
ARVO Annual Meeting Abstract  |   June 2021
Parthanatos operates intraocularly during murine cytomegalovirus (MCMV) retinitis in mice with acquired immunodeficiency syndrome (MAIDS): An investigation of possible cell-type specificity
Author Affiliations & Notes
  • Jay Oh
    Biology, Georgia State University, Atlanta, Georgia, United States
  • Richard D Dix
    Biology, Georgia State University, Atlanta, Georgia, United States
    Ophthalmology, Emory University School of Medicine, Atlanta, Georgia, United States
  • Footnotes
    Commercial Relationships   Jay Oh, None; Richard Dix, None
  • Footnotes
    Support  NIH/NEI Grant RO1 EY024630, NIH/NEI Core Grant P30 EY006360, NIH/NEI T32 EY007092, Fight for Sight, Inc., Molecular Basis of Diseases Fellowship from Georgia State University.
Investigative Ophthalmology & Visual Science June 2021, Vol.62, 1953. doi:
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      Jay Oh, Richard D Dix; Parthanatos operates intraocularly during murine cytomegalovirus (MCMV) retinitis in mice with acquired immunodeficiency syndrome (MAIDS): An investigation of possible cell-type specificity. Invest. Ophthalmol. Vis. Sci. 2021;62(8):1953.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Parthanatos is a caspase-independent cell death pathway whose contributions to retinal tissue destruction during the progression of AIDS-related human cytomegalovirus retinitis have yet to be explored. We have previously shown that key parthanatos proteins are stimulated within MCMV-infected eyes of retinitis-susceptible mice with MAIDS of 10-weeks duration (MAIDS-10) (Oh et al, J Med Virol, 2019) but not within MCMV-infected eyes of retinitis-resistant mice with MAIDS of 4-weeks duration (MAIDS-4) (unpublished data). That stimulation of parthantos during MCMV-related MCMV retinitis might be cell-type specific prompted us to perform a pilot study to test this hypothesis using mouse embryo fibroblasts (MEF) and mouse lung fibroblasts (MLg), both non-ocular cells known to be susceptible to MCMV infection.

Methods : Monolayers of MEF and MLg were inoculated with MCMV (moi = 1) or maintenance medium (control). Cell lysates of MCMV-infected and mock-infected cells were harvested at 24, 36, and 72 hrs after infection and subjected to western blot analysis for detection of parthantos-associated PARP-1, PAR, and PARG proteins.

Results : Whereas MCMV-infected MEF showed stimulation of PAR and PARG when compared with mock-infected cells, stimulation of these two key parthanatos-associated proteins was not observed within MCMV-infected MLg at all times investigated. In comparison, PARP-1 protein showed significantly reduced stimulation within MCMV-infected MLg when compared with MCMV-infected MEF, but equivalent amounts of PARP-1 protein production were also observed within mock-infected MEF at all times investigated suggesting its stimulation was due to trauma associated with the inoculation procedure.

Conclusions : Our findings support the hypothesis that stimulation of the parthantos cell death pathway during MCMV infection is cell-type specific. This proof-of-principle finding supports further investigations by us to determine if parthanatos operates within individual cell types of the retina during MAIDS-related MCMV retinitis with a focus on the retinal pigmented epithelium.

This is a 2021 ARVO Annual Meeting abstract.

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