June 2021
Volume 62, Issue 8
Open Access
ARVO Annual Meeting Abstract  |   June 2021
Saccade-Pursuit coordination during ocular tracking across different impairment sources
Author Affiliations & Notes
  • Terence L. Tyson
    Human Systems Integration Division, NASA Ames Research Center, Moffett Field, California, United States
  • Erin E. Flynn-Evans
    Human Systems Integration Division, NASA Ames Research Center, Moffett Field, California, United States
  • Leland S. Stone
    Human Systems Integration Division, NASA Ames Research Center, Moffett Field, California, United States
  • Footnotes
    Commercial Relationships   Terence Tyson, None; Erin Flynn-Evans, None; Leland Stone, NASA-held US patents Nos 9,730,582/10,420,465/10,463,249 awarded 8/2017, 9/2019, and 10/2019, respectively, but author has no direct role in any commercialization. (P)
  • Footnotes
    Support  NASA's Human Research Program
Investigative Ophthalmology & Visual Science June 2021, Vol.62, 2390. doi:
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    • Get Citation

      Terence L. Tyson, Erin E. Flynn-Evans, Leland S. Stone; Saccade-Pursuit coordination during ocular tracking across different impairment sources. Invest. Ophthalmol. Vis. Sci. 2021;62(8):2390.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Sleep loss (Stone et al., 2019, doi: 10.1113/JP277779) and alcohol (Tyson et al., 2020, doi: 10.1113/JP280395) have been shown to impair smooth pursuit and its underlying visual motion processing in humans. This study examines the saccadic compensation for this poor pursuit.

Methods : Using an established behavioral ocular-tracking paradigm (Liston & Stone, 2014, doi: 10.1167/14.14.12), we examined the dose-response of ground lost (pursuit deficit integrated across our 300-ms steady-state tracking interval) and ground gained (increased saccadic response harnessed to compensate) across three separate studies – acute low-dose alcohol administration (LDA; n = 16 subjects), acute sleep loss (ASL; n = 12), and chronic sleep restriction (CSR; n = 12). We computed dose-responses as the linear regression slopes of ground lost and gained across treatment dose (% blood alcohol concentration [BAC] or hours awake). For the CSR study, we computed the mean effect for a single dose (5-hours nightly sleep for 1 week).

Results : For LDA, there was significantly increased ground lost (P < 0.001) and gained (P < 0.001) with increased %BAC. In addition, the dose-responses were not significantly different (P = 0.35), indicating effectively complete saccadic compensation due to significant increases in both saccadic rate (P < 0.05) and amplitude (P < 0.001). For ASL, there was significantly increased ground lost with time awake (P < 0.01), however ground gained was significantly lower (P < 0.001), indicating, at best, incomplete compensation due to a significant increase in saccadic rate (P < 0.001) but not amplitude (P = 0.10). With CSR, pursuit was again significantly impaired (P < 0.05), with saccadic rate significantly increased (P < 0.05) but, surprisingly, amplitude was significantly decreased (P < 0.05), effectively eliminating ground gained (P = 0.90).

Conclusions : Our analyses show that LDA, ASL, and CSR affect tracking differently, suggesting the involvement of different brain pathways. With LDA, the effect appears largely due to the cortical impairment of visual motion processing with largely healthy brainstem and mid-brain responses (driving effective saccadic compensation). ASL and CSR however appear to affect both cortical and sub-cortical pathways, with at best partial saccadic compensation. Lastly, CSR is associated with an additional compromise due to a maladaptive decrease in saccade amplitude.

This is a 2021 ARVO Annual Meeting abstract.

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