June 2022
Volume 63, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2022
SZN-413, a monospecific Fzd4 Agonist as a Potential Novel Therapeutic for the Treatment of Diabetic Retinopathy
Author Affiliations & Notes
  • Huy Nguyen
    Surrozen, California, United States
  • Sungjin Lee
    Surrozen, California, United States
  • Elizabeth Whisler
    Surrozen, California, United States
  • Kelsey Logas
    Surrozen, California, United States
  • Hui Chen
    Surrozen, California, United States
  • Partha Sampathkumar
    Surrozen, California, United States
  • Jay Ye
    Surrozen, California, United States
  • Nick Suen
    Surrozen, California, United States
  • Yang Li
    Surrozen, California, United States
  • Wen-Chen Yeh
    Surrozen, California, United States
  • Footnotes
    Commercial Relationships   Huy Nguyen Surrozen, Code E (Employment); Sungjin Lee Surrozen, Code E (Employment); Elizabeth Whisler Surrozen, Code E (Employment); Kelsey Logas Surrozen, Code E (Employment); Hui Chen Surrozen, Code E (Employment); Partha Sampathkumar Surrozen, Code E (Employment); Jay Ye Surrozen, Code E (Employment); Nick Suen Surrozen, Code E (Employment); Yang Li Surrozen, Code E (Employment); Wen-Chen Yeh Surrozen, Code E (Employment)
  • Footnotes
    Support  None
Investigative Ophthalmology & Visual Science June 2022, Vol.63, 668 – F0122. doi:
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      Huy Nguyen, Sungjin Lee, Elizabeth Whisler, Kelsey Logas, Hui Chen, Partha Sampathkumar, Jay Ye, Nick Suen, Yang Li, Wen-Chen Yeh; SZN-413, a monospecific Fzd4 Agonist as a Potential Novel Therapeutic for the Treatment of Diabetic Retinopathy. Invest. Ophthalmol. Vis. Sci. 2022;63(7):668 – F0122.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Norrin/Fzd4 signaling is indispensable for retinal vascular development and vessel function in humans and rodent models. These studies examined whether a novel Norrin mimetic could promote the regeneration of damaged blood vessels and their functions in diabetic retinopathy animal models.

Methods : We generated an antibody-based bi-specific Norrin mimetic (SZN-413) that targets Fzd4 and low-density lipoprotein receptor-related proteins and evaluated its effects on damaged retinal vessels in mice and rabbit models. In an oxygen-induced retinopathy (OIR) mouse model, SZN-413 was delivered intravitreally (IVT) and the avascular (AV) area and neovascularization (NV) area were measured 5 days later. Furthermore, the impact on vascular leakage by SZN-413 was examined in a VEGF-induced retinal vascular leakage rabbit model, in which the level of fluorescein leakage was measured 3 days after IVT delivery of VEGF together with SZN-413 or vehicle. ANOVA was used for statistical analysis.

Results : Results: In the OIR mouse model, nanogram quantities of SZN-413 significantly reduced the NV area size (p<0.001) to a level comparable to the group treated with 60 ug aflibercept. SZN-413 also showed a dramatic reduction in AV area size compared to vehicle (p<0.001) and compared to aflibercept (p<0.01). In the VEGF-induced retinal vascular leakage rabbit model, SZN-413 significantly reduced retinal vascular leakage by 78%, compared to the vehicle-treated group (p<0.01). No observable abnormalities were detected in ocular exams in these studies.

Conclusions : The novel Norrin mimetic, SZN-413, demonstrated anti-NV properties and reduced the avascular area in OIR mice, and reduced VEGF-driven retinal vascular leakage in rabbits. The results strongly suggests that the pathological cellular responses can be modulated by SZN-413, with possible therapeutic implications for diabetic retinopathy.

This abstract was presented at the 2022 ARVO Annual Meeting, held in Denver, CO, May 1-4, 2022, and virtually.

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