June 2022
Volume 63, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2022
D and E series Specialized Pro-Resolving Mediators Inhibit Capsaicin-induced Calcium Influx in Rat Trigeminal Ganglionic Neurons
Author Affiliations & Notes
  • Jeffrey Bair
    Schepens Eye Research Institute of Massachusetts Eye and Ear, Boston, Massachusetts, United States
  • Amy E Ross
    Schepens Eye Research Institute of Massachusetts Eye and Ear, Boston, Massachusetts, United States
  • Darlene A Dartt
    Schepens Eye Research Institute of Massachusetts Eye and Ear, Boston, Massachusetts, United States
  • Joeseph Ciolino
    Schepens Eye Research Institute of Massachusetts Eye and Ear, Boston, Massachusetts, United States
  • Charles N Serhan
    Center for Experimental Therapeutics and Reperfusion Injury, Department of Anesthesia, Brigham and Women's Hospital, Boston, Massachusetts, United States
  • Footnotes
    Commercial Relationships   Jeffrey Bair None; Amy Ross None; Darlene Dartt None; Joeseph Ciolino None; Charles Serhan None
  • Footnotes
    Support  Financial support was provided by the Kohli Prism Fund
Investigative Ophthalmology & Visual Science June 2022, Vol.63, 1221 – A0221. doi:
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      Jeffrey Bair, Amy E Ross, Darlene A Dartt, Joeseph Ciolino, Charles N Serhan; D and E series Specialized Pro-Resolving Mediators Inhibit Capsaicin-induced Calcium Influx in Rat Trigeminal Ganglionic Neurons. Invest. Ophthalmol. Vis. Sci. 2022;63(7):1221 – A0221.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : E-series resolvins (Rv), derived from the n-fatty acid, eicosapentaenoic acid (EPA), and the D-series Rvs, protectins, and maresins all derived from the n-fatty acid docosahexaenoic acid (DHA), comprise three categories of specialized pro-resolving mediators (SPMs) that actively resolve inflammation in disease. The purpose of this study was to determine if one or more of these SPM families can attenuate the capsaicin-evoked activity of rat trigeminal ganglion neurons indicating a decrease in corneal pain.

Methods : Rat trigeminal ganglia (TG) were extracted and dissociated first in a solution of papain in HBSS for 20 minutes before transferred to a solution of collagenase and dispase for a further 20 minutes. The dissociated cells were then separated using a percoll density gradient. The TG neurons were isolated and plated onto poly-L-lysine and laminin coated glass-bottom dishes and left to grow overnight. The following day, cells were washed with Krebs Ringer Buffer (KRB) and loaded with the calcium-sensitive fluorescent dye fura2. An increase in intracellular [Ca2+] indicated activation of the neuron and pain in the nerve endings. Thirty minutes prior to the experiment cells were treated with one of the following: RvD1, RvD2, RvD3, RvD4, RvD5, or RvE1 at concentrations between 10-11M and 10-8M. The cells were then stimulated with capsaicin (10-7M) and intracellular [Ca2+] were recorded over time.

Results : Pre-treatment with RvD1 10-8M reduced the magnitude of the capsaicin-induced calcium influx by an average of 36.03% (N=7 p=0.013). RvD2 10-11, 10-10, 10-9, and 10-8M reduced the magnitude of the response by an average of 49.29%, 49.57%, 35.64% and 63.58% respectively (N=7, p= 0.001, 0.002, 0.001, and 0.001 respectively). RvD3 10-8M inhibited the response by an average of 25.05% (N=4, p =0.003). RvD5 10-9M inhibited the response by an average of 59.93% (N=4, p= 0.003). RvE1 10-10M significantly reduced intracellular Ca2+ influx following capsaicin administration by an average of 19.32% (N=4, p=0.042).

Conclusions : The SPMs RvD1, RvD2, RvD3, RvD5, and RvE1 significantly decrease capsaicin-evoked intracellular [Ca2+] influx in rat TG, with RvD2 being the most potent SPM treatment could decrease ocular surface pain.

This abstract was presented at the 2022 ARVO Annual Meeting, held in Denver, CO, May 1-4, 2022, and virtually.

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