June 2022
Volume 63, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2022
Nrf2 Knockout Mice Exhibit High Glycemic Index Diet Associated Transcriptomic Alterations in Liver and Retina
Author Affiliations & Notes
  • Daniel C Brock
    National Eye Institute, Bethesda, Maryland, United States
  • Sheldon Rowan
    Tufts University, Boston, Massachusetts, United States
  • Anupam Kumar Mondal
    National Eye Institute, Bethesda, Maryland, United States
  • Milton English
    National Eye Institute, Bethesda, Maryland, United States
  • Allen Taylor
    Tufts University, Boston, Massachusetts, United States
  • Anand Swaroop
    National Eye Institute, Bethesda, Maryland, United States
  • Footnotes
    Commercial Relationships   Daniel Brock None; Sheldon Rowan None; Anupam Mondal None; Milton English None; Allen Taylor None; Anand Swaroop None
  • Footnotes
    Support  NEI IRP ZIAEY000450 and ZIAEY000546 (to Anand Swaroop.); NIH RO1EY028559, RO1EY026979, USDA 8050-51000-089-01S (to Allen Taylor).
Investigative Ophthalmology & Visual Science June 2022, Vol.63, 1929 – F0347. doi:
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      Daniel C Brock, Sheldon Rowan, Anupam Kumar Mondal, Milton English, Allen Taylor, Anand Swaroop; Nrf2 Knockout Mice Exhibit High Glycemic Index Diet Associated Transcriptomic Alterations in Liver and Retina. Invest. Ophthalmol. Vis. Sci. 2022;63(7):1929 – F0347.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Dietary glycemic index (GI) indicates how fast glucose from dietary carbohydrates enters the bloodstream after consuming a food item. Dietary GI has been found as a risk factor for developing blinding diseases, such as age-related macular degeneration and cataracts. The retinas of mice fed a high GI diet for 12 months show photoreceptor loss, which is accelerated in Nrf2 knockout mice. To assess the molecular consequences of GI in WT and Nrf2-/- mice, we have evaluated transcriptomic changes in the retinas and livers after exposure to a high (HGI) or low glycemic (LGI) diet.

Methods : C57BL/6 (WT) and Nrf2-/- mice were aged from 6 to 18 months while feeding on an LGI diet of 70% amylose + 30% amylopectin or an HGI diet of 100% amylopectin. RNA from retinas and livers was used for RNA-seq. Bioinformatic analyses involved the Bioconductor pipeline of edgeR and limma. Differential analysis was set at two fold change and p-value of 0.05, before pathway annotation with gProfiler.

Results : Principal component analysis showed distinct genotype-based clustering among WT and Nrf2-/- livers. WT HGI mice displayed higher expression of glutathione pathway genes, such as glutathione S-transferase, relative to LGI. Notably, glutathione genes were reduced by 2-6 fold in Nrf2-/- livers, regardless of GI, indicating gene-diet interactions. The expression of retinol metabolism genes, including aldehyde dehydrogenase and UDP glucuronosyltransferase, were also reduced in Nrf2-/- livers. Mitochondrial OXPHOS genes, involved in complexes 1 and 5, as well as late endosomal/lysosomal autophagy genes, were upregulated in WT HGI livers but did not show any response to GI in Nrf2-/- livers. Retinal transcriptome analysis is in progress.

Conclusions : The interaction between genetics and dietary GI in WT and Nrf2-/- mice can help explain the differences in phenotype and disease susceptibility. We reason that Nrf2-/- animals experience reduced ability to respond to glycemia-induced stress due to attenuated antioxidant response systems. This leads to a lowered ability to sanitize the cell of damaging molecules. Retinal transcriptomic analysis would reveal a pathmechanistic relationship between GI, genotype, and retinal degeneration.

This abstract was presented at the 2022 ARVO Annual Meeting, held in Denver, CO, May 1-4, 2022, and virtually.

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