June 2022
Volume 63, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2022
Defective photoreceptor-to-bipolar cell synaptic transmission in novel knock-in mouse models of RP59
Author Affiliations & Notes
  • Mai N Nguyen
    Optometry and Vision Science, The University of Alabama at Birmingham, Birmingham, Alabama, United States
  • Dibyendu Chakraborty
    Optometry and Vision Science, The University of Alabama at Birmingham, Birmingham, Alabama, United States
  • Steven J Fliesler
    Ophthalmology, Biochemistry, and Neuroscience Graduate Program, SUNY The State University of New York, Buffalo, New York, United States
    Research Service, VA Western New York Healthcare System, Buffalo, New York, United States
  • Steven J Pittler
    Optometry and Vision Science, The University of Alabama at Birmingham, Birmingham, Alabama, United States
  • Footnotes
    Commercial Relationships   Mai Nguyen None; Dibyendu Chakraborty None; Steven Fliesler None; Steven Pittler None
  • Footnotes
    Support  NIH/NEI (F31 EY032764; MNN), NIH/NEI (1 R01 EY029341; SJP, SJF), P30 EY003039 (SJP), NIH/NCATS (1UL1 TR001412; SJF), VAWNYHS facilities and resources (SJF), VA Research Career Scientist Award/BLR&D I K6 BX005787 (SJF)
Investigative Ophthalmology & Visual Science June 2022, Vol.63, 1896 – A0042. doi:
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    • Get Citation

      Mai N Nguyen, Dibyendu Chakraborty, Steven J Fliesler, Steven J Pittler; Defective photoreceptor-to-bipolar cell synaptic transmission in novel knock-in mouse models of RP59. Invest. Ophthalmol. Vis. Sci. 2022;63(7):1896 – A0042.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : RP59 (OMIM# 613861) is caused by mutations in the dehydrodolichyl diphosphate synthase (DHDDS) gene. We previously reported that knock-in mice with K42E and T206A DHDDS mutations do not exhibit overt retinal degeneration, but exhibit a “negative ERG” [normal a-wave and reduced (scotopic and photopic) b-wave amplitudes]. Here, we describe further characterization of two RP59 mouse models.

Methods : DhddsT206A/T206A and DhddsT206A/K42E knock-in mice (Nguyen et al., ARVO 2021) vs. age-matched WT (Dhdds+/+) mice were assessed by quantitative retinal morphometry (SD-OCT) and electroretinography (ERG; 500 nm LED stimulus). Visual acuity (VA; spatial frequency) was assessed by OKR under scotopic and photopic conditions. Statistical significance (Student’s t-test) threshold was p≤0.05 (N≥3 SD-OCT, N≥8 ERG, N≥3 OKR).

Results : From PN 1- to PN 12-mo, neural retina full thickness declined <5% (p≤0.05), while INL thickness declined by 27% in DhddsT206A/T206A and 30% in DhddsT206A/K42E (p≤0.01), vs. WT mice. ERG b-to-a-wave amplitude (b/a) ratios declined (at 3-mo intervals) over the same time. At PN 12-mo: scotopic b/a ratios were reduced by ~25% in DhddsT206A/T206A and by ~35% in DhddsT206A/K42E mice, vs. WT values (p≤0.01). Photopic b/a ratios were also declined by ~33% in DhddsT206A/T206A and by ~41% in DhddsT206A/K42E mice, vs. WT values (p≤0.01). ERG c-wave amplitudes were like WT at PN 6-mo; however, at PN 12-mo, c-wave amplitudes were reduced by ~35% in both mutants, vs. WT mice (p≤0.01), and ERG d-wave amplitudes were reduced by ~35% in DhddsT206A/K42E mice, vs. WT values (p≤0.05). Scotopic VA was reduced by ~15% and ~10%, respectively, in DhddsT206A/T206A and DhddsT206A/K42E mice, and photopic VA was reduced by ~15% and ~20.5%, respectively, in DhddsT206A/T206A and DhddsT206A/K42E mice, vs. WT values.

Conclusions : These results are consistent with defective rod- and cone-to-bipolar cell synaptic transmission (normal a-wave, reduced b- and d-waves) and compromised RPE and/or Müller cell responses (reduced c-wave) in these Dhdds mouse mutants.

This abstract was presented at the 2022 ARVO Annual Meeting, held in Denver, CO, May 1-4, 2022, and virtually.

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