June 2022
Volume 63, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2022
Inhibition of HIF-1α mRNA could regulate monocyte functions and could inhibit neovascularization in a mouse model of proliferative retinopathy
Author Affiliations & Notes
  • MD Imam Uddin
    Ophthalmology, Vanderbilt University School of Medicine, Nashville, Tennessee, United States
    Biomedical Engineering, Vanderbilt University, Nashville, Tennessee, United States
  • Rita Atalor
    Ophthalmology, Vanderbilt University School of Medicine, Nashville, Tennessee, United States
  • Footnotes
    Commercial Relationships   MD Imam Uddin Vanderbilt University , Code P (Patent); Rita Atalor None
  • Footnotes
    Support  (1) R01EY029693-01. (2) R01EY023397-07
Investigative Ophthalmology & Visual Science June 2022, Vol.63, 2831 – A0347. doi:
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    • Get Citation

      MD Imam Uddin, Rita Atalor; Inhibition of HIF-1α mRNA could regulate monocyte functions and could inhibit neovascularization in a mouse model of proliferative retinopathy. Invest. Ophthalmol. Vis. Sci. 2022;63(7):2831 – A0347.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Bone marrow-derived activated monocytes and progenitor cells migrate to the retina in response to inflammation and neovascularization. Hypoxia-inducible factor-1α (HIF-1α) has been shown to contribute to the pathogenesis of neovascularization. However, contribution of monocyte-derived macrophages to neovascularization is largely unknown. We describe here the synthesis of a new hybrid nanoparticle for targeted delivery and gene silencing in activated monocytes that are associated with pathological neovascularization.

Methods : Single-cell RNAseq data analysis were performed to characterize HIF-1α mRNA expression in activated monocytes in mouse oxygen-induced retinopathy (OIR). HIF-1α targeted AS-shRNA-lipids were synthesized by conjugating diacyl-lipids to anti-sense short hairpin RNA with an anti-sense sequence complimentary to HIF-1α mRNA. The short hairpin RNA compounds were stabilized using 2’-O-methyl-protected ribonucleotides allowing in vivo delivery of AS-shRNA-lipid to the activated monocytes that are associated with neovascularization.

Results : HIF-1α mRNA expression was associated with MRC-1 positive activated monocytes. In addition, HIF-1α mRNA was induced in monocytes by exposing the cells to hypoxia, and the expression was inhibited by AS-HIF-1α-shRNA-lipid. CD14 mRNA expression was significantly elevated in monocytes that were exposed to hypoxia, suggesting monocyte activation. AS-HIF-1α-shRNA-lipid could significantly inhibit this activation. In addition, significant reduction of neovascularization was achieved in mouse OIR after intraperitoneal injection of AS-shRNA-lipids.

Conclusions : Inflammation and hypoxia could activate monocyte functions and inhibition of HIF-1α mRNA could recover these cells from activation. In summary, we have developed a novel method for targeted delivery and inhibit HIF-1α mRNA in activated monocytes in living ocular tissues using diacyl-lipid-conjugates of antisense short hairpin RNA (AS-shRNA-lipid) designed to target HIF-1α mRNA. These conjugates are readily internalized by activated monocytes that are associated with neovascularization in the living retina. These findings may provide a framework for a novel strategy to inhibit retinal neovascularization.

Keywords: targeted delivery, activated monocytes, gene therapy, short-hairpin RNA, neovascularization.

This abstract was presented at the 2022 ARVO Annual Meeting, held in Denver, CO, May 1-4, 2022, and virtually.

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