June 2022
Volume 63, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2022
Multiscale imaging of mouse corneal endothelial cell damage induced by elevated intraocular pressure
Author Affiliations & Notes
  • Junghun Kweon
    Biomedical Engineering, Northwestern University, Evanston, Illinois, United States
  • Yang Zhang
    Biomedical Engineering, Northwestern University, Evanston, Illinois, United States
  • Benjamin Samuel brenner
    Biomedical Engineering, Northwestern University, Evanston, Illinois, United States
  • Raymond Fang
    Biomedical Engineering, Northwestern University, Evanston, Illinois, United States
  • Cheng Sun
    Mechanical Engineering, Northwestern University, Evanston, Illinois, United States
  • Jeffrey L Goldberg
    Byers Eye Institute, Stanford University, Stanford, California, United States
  • Hao Zhang
    Biomedical Engineering, Northwestern University, Evanston, Illinois, United States
  • Footnotes
    Commercial Relationships   Junghun Kweon None; Yang Zhang None; Benjamin brenner None; Raymond Fang None; Cheng Sun None; Jeffrey Goldberg Opticent Health, Code I (Personal Financial Interest); Hao Zhang Opticent Health, Code I (Personal Financial Interest)
  • Footnotes
    Support  None
Investigative Ophthalmology & Visual Science June 2022, Vol.63, 2741 – A0230. doi:
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      Junghun Kweon, Yang Zhang, Benjamin Samuel brenner, Raymond Fang, Cheng Sun, Jeffrey L Goldberg, Hao Zhang; Multiscale imaging of mouse corneal endothelial cell damage induced by elevated intraocular pressure. Invest. Ophthalmol. Vis. Sci. 2022;63(7):2741 – A0230.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Hexagonal-shaped corneal endothelial cells (CECs) are essential in maintaining corneal transparency for clear vision. In glaucoma patients, abnormal CEC function caused by high intraocular pressure (IOP) can contribute to vision loss. We developed a multi-scale imaging workflow to determine whether we could identify morphological and structural variations at at the nanoscopic level using a mouse model of high IOP induced by anterior chamber cannulation

Methods : We cannulated the left eyes of wild-type (WT) mice using anterior chamber puncture, and controlled the IOP to be 40 mmHg by adjusting the height of the syringe; right eyes served as controls. We first used a custom visible-light optical coherence tomography (vis-OCT) to monitor the overall corneal damage in real-time. After one hour of hypertension treatment, we sacrificed the mice, isolated their corneas, and processed one half for single-molecule localization microscopy (SMLM) and the other half for scanning electron microscopy (SEM)

Results : The vis-OCT images of control corneas showed a typical normal stroma (Fig. 1A). Increased corneal thickness was observed (Fig. 1B) with typical edematous appearance after maintaining 40 mmHg IOP for one hour. SMLM images showed the typical hexagonal structures of CECs in controls (Fig. 1C). However, after hypertension treatment, the intercellular tight junction structures labeled by ZO-1 became distorted dramatically, showing variations including extended filaments (Fig. 1E) or discontinuous and thinner junctional structures (Fig. 1D) between CECs. Using SEM, we identified similar hexagonal structures of CECs as showed in SMLM (Fig. 1F). Meanwhile, we observed the junctional structural variations on the surface level of CECs in hypertensive corneas, validating SMLM imaging results (Fig. 1G)

Conclusions : We imaged corneal flat-mounts to reveal nanoscopic variations in CECs in hypertension mouse models using vis-OCT, SMLM, and SEM. Such disruptions of the intercellular junctional structure of CECs in corneas may contribute to the loss of transparency and vision damage in glaucoma patients with high IOP

This abstract was presented at the 2022 ARVO Annual Meeting, held in Denver, CO, May 1-4, 2022, and virtually.

 

Figure 1. (A-B) Vis-OCT images of mouse cornea before (A) and after (B) maintaining 40 mmHg IOP for 1 h; (C-E) SMLM images of tight junctions in ZO-1-labeled CECs in corneal flat-mounts of control eye (C) and hypertensive eyes (D, E) ; (F-G) SEM images of a control eye (F) and hypertensive eye (G)

Figure 1. (A-B) Vis-OCT images of mouse cornea before (A) and after (B) maintaining 40 mmHg IOP for 1 h; (C-E) SMLM images of tight junctions in ZO-1-labeled CECs in corneal flat-mounts of control eye (C) and hypertensive eyes (D, E) ; (F-G) SEM images of a control eye (F) and hypertensive eye (G)

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