June 2023
Volume 64, Issue 8
Open Access
ARVO Annual Meeting Abstract  |   June 2023
Inhibition of pterygium cell fibrosis by the Rho kinase inhibitor
Author Affiliations & Notes
  • Jiannong Dai
    Ophthalmalogy, Indiana University Department of Ophthalmology, Indianapolis, Indiana, United States
  • naga rayana
    Tufts University School of Medicine, Boston, Massachusetts, United States
  • Michael Peng
    Ophthalmalogy, Indiana University Department of Ophthalmology, Indianapolis, Indiana, United States
  • Chenna Kesavulu Sugali
    Ophthalmalogy, Indiana University Department of Ophthalmology, Indianapolis, Indiana, United States
  • Shaohui Liu
    Ophthalmalogy, Indiana University Department of Ophthalmology, Indianapolis, Indiana, United States
  • Weiming Mao
    Ophthalmalogy, Indiana University Department of Ophthalmology, Indianapolis, Indiana, United States
  • Footnotes
    Commercial Relationships   Jiannong Dai None; naga rayana None; Michael Peng None; Chenna Kesavulu Sugali None; Shaohui Liu None; Weiming Mao None
  • Footnotes
    Support  This study was supported by the National Institute of Health/National Eye Institute Award Numbers R01EY026962 (WM), R01EY031700 (WM), and a challenge grant from Research to Prevent Blindness (Department of Ophthalmology, Indiana University School of Medicine).
Investigative Ophthalmology & Visual Science June 2023, Vol.64, 1395. doi:
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    • Get Citation

      Jiannong Dai, naga rayana, Michael Peng, Chenna Kesavulu Sugali, Shaohui Liu, Weiming Mao; Inhibition of pterygium cell fibrosis by the Rho kinase inhibitor. Invest. Ophthalmol. Vis. Sci. 2023;64(8):1395.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Pterygium is an ocular disease in which the conjunctival tissue grows onto the cornea. When the pterygium tissue reaches the pupillary region, vision will be affected. Currently, surgical removal is the only effective treatment. However, the recurrence rate of pterygium after surgery can be high. Although mitomycin C has been used to decrease recurrence, it is highly toxic to ocular surface tissues. Rho kinase inhibitors are used for lowering intraocular pressure and have been reported to inhibit extracellular matrix deposition in the trabecular meshwork. Therefore, we determined if the less toxic Rho kinase inhibitor can be used to decrease pterygium cell growth and migration.

Methods : The study was approved by Indiana University School of Medicine IRB. After obtaining inform consent from the patient, we collected surgically removed pterygium tissues. The tissues were dissected and used directly for migration assays or cultured for primary pterygium cell strain establishment. Primary human pterygium cells were grown in the Opti-MEM medium supplemented with 5% FBS and antibiotics, and only cells with early passage numbers (<p7) were used. The cells were treated with DMSO or the Rho kinase inhibitor Y27632 at 10 or 20 µM. The cells were studied using cell migration assay, scratch assay, viability assay, and immunostaining.

Results : We found that 10uM Y27632 significantly deceased pterygium cell viability (P<0.05; N=3). It also slowed down pterygium cell migration and/or proliferation from pterygium tissues as well as in scratch assays. Y27632 decreased the expression of fibrosis markers including fibronectin, collegen-1 and alpha-smooth muscle actin.

Conclusions : Conclusion: We found that the Rho kinase inhibitor Y27632 decreased cell migration, delayed wound healing, and decreased expression of myofibroblast marker. We believe that Rho kinases inhibitors are a potential post-surgical treatment to prevent or slow down pterygium recurrence.

This abstract was presented at the 2023 ARVO Annual Meeting, held in New Orleans, LA, April 23-27, 2023.

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