June 2023
Volume 64, Issue 8
Open Access
ARVO Annual Meeting Abstract  |   June 2023
Retinoic acid weakens synaptic transmission between ON cone bipolar cells and RGCs in mouse models of retinitis pigmentosa
Author Affiliations & Notes
  • Logan Ganzen
    University of California Berkeley, Berkeley, California, United States
  • Shubhash Chandra Yadav
    University of California Berkeley, Berkeley, California, United States
  • Mingxiao Wei
    University of California Berkeley, Berkeley, California, United States
  • Scott Nawy
    University of California Berkeley, Berkeley, California, United States
  • Richard Kramer
    University of California Berkeley, Berkeley, California, United States
  • Footnotes
    Commercial Relationships   Logan Ganzen None; Shubhash Chandra Yadav None; Mingxiao Wei None; Scott Nawy None; Richard Kramer None
  • Footnotes
    Support  This work was supported by grants from the NIH (R01EY024334 and P30EY003176) and the Foundation Fighting Blindness Gund-Harrington Award
Investigative Ophthalmology & Visual Science June 2023, Vol.64, 3212. doi:
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    • Get Citation

      Logan Ganzen, Shubhash Chandra Yadav, Mingxiao Wei, Scott Nawy, Richard Kramer; Retinoic acid weakens synaptic transmission between ON cone bipolar cells and RGCs in mouse models of retinitis pigmentosa. Invest. Ophthalmol. Vis. Sci. 2023;64(8):3212.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : While rods and cones degenerate in retinitis pigmentosa and age-related macular degeneration, retinal ganglion cells (RGCs) and bipolar cells survive, providing multiple possible substrates for vision restoration. However, RGCs undergo physiological remodeling, exhibiting hyperactive firing. Here we ask if PR degeneration also affects the synapse between Type 6 ON-cone bipolar cells (CBC6) and ON αRGCs.

Methods : We used rd1 (P60) and rd10 (P90) mice which exhibit photoreceptor degeneration. CBC6 cells were identified through selective genetic expression of ChR2/EYFP fusion protein. To measure voltage-gated voltage-gated Ca2+ current (ICa), we employed whole-cell patch clamp on CBC6 cells in slices. To measure synaptic transmission, we optogenetically stimulated CBC6 in flat-mount retinas and recorded excitatory post-synaptic currents (EPSCs) in ON αRGCs. To measure synaptic density, we visualized synaptic ribbons in CBC6 cells with immunohistological staining with anti-CTBP2 (RIBEYE).To block retinoic acid (RA) signaling, we intravitreally injected the RA receptor inhibitor BMS 493.

Results : RIBEYE staining showed no change in the number or density of CBC6 synaptic ribbons adjacent to dye-filled ON αRGCs. However, the intrinsic ICa of CBC6 cells was 70% smaller in rd1 than in wild-type (WT) mice. Optogenetically evoked EPSCs recorded in ON αRGCs were ~75% and ~50% smaller in rd1 and rd10 respectively. Paired-pulse stimulation caused synaptic depression in WT but none in rd1 and rd10 retinas. RA has been implicated as a key trigger of retinal remodeling. Spatial representation of multiple CBC6 inputs onto an individual ON αRGC was unchanged in rd1 retinas, but temporal filtering was dramatically altered. Blocking RA signaling with BMS 493 restored both CBC6 ICa and synaptic transmission in rd1 mice.

Conclusions : Photoreceptor degeneration dramatically alters signaling in the downstream ON pathway. Bipolar cells have less calcium current thereby reducing synaptic transmission. These changes will attenuate signals initiated by surviving photoreceptors or by vision restoration tools such as optogenetics, optopharmocology, stem cell therapy, and subretinal implants. Fortunately, blocking RA signaling may suppress remodeling, mitigating these vision-corrupting effects.

This abstract was presented at the 2023 ARVO Annual Meeting, held in New Orleans, LA, April 23-27, 2023.

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