June 2023
Volume 64, Issue 8
Open Access
ARVO Annual Meeting Abstract  |   June 2023
A multiplexed single-cell sequencing based screen identifies compounds that increase the neurogenic potential of Muller glia in the adult mouse retina.
Author Affiliations & Notes
  • Levi Todd
    Biological Structure, University of Washington, Seattle, Washington, United States
  • Amy Tresenrider
    Genome Sciences, University of Washington, Seattle, Washington, United States
  • Marcus Hooper
    Biological Structure, University of Washington, Seattle, Washington, United States
  • Faith Kierney
    Biological Structure, University of Washington, Seattle, Washington, United States
  • Cole Trapnell
    Genome Sciences, University of Washington, Seattle, Washington, United States
  • Thomas A Reh
    Biological Structure, University of Washington, Seattle, Washington, United States
  • Footnotes
    Commercial Relationships   Levi Todd None; Amy Tresenrider None; Marcus Hooper None; Faith Kierney None; Cole Trapnell None; Thomas Reh None
  • Footnotes
    Support  NEI K99EY033402 to L.T., R01EY021482 to T.A.R., NEI F32 EY032331 to A.T.
Investigative Ophthalmology & Visual Science June 2023, Vol.64, 4344. doi:
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      Levi Todd, Amy Tresenrider, Marcus Hooper, Faith Kierney, Cole Trapnell, Thomas A Reh; A multiplexed single-cell sequencing based screen identifies compounds that increase the neurogenic potential of Muller glia in the adult mouse retina.. Invest. Ophthalmol. Vis. Sci. 2023;64(8):4344.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Neurodegeneration in the mammalian retina results in permanent blindness. Significant progress has been made in stimulating Muller glia (MG) to regenerate functional neurons in the adult mouse by transgenic expression of the transcription factor Ascl1. While these results showed that MG can serve as an endogenous source of neuronal replacement, the efficacy of this process is limited: only 25% of the MG generate neurons.

To uncover molecules that enhance neurogenic reprogramming of MG, we combined our in vitro MG reprogramming paradigm with sci-Plex to test a library of 96 compounds for their ability to promote neurogenesis using scRNA-seq as a read out. We identified molecules that increase neurogenesis from MG the adult mouse retina.

Methods : sci-Plex uses combinatorial indexing of nuclei from individual wells “hashed” with DNA oligos that serve as sample-specific barcodes. For in vitro screening, cultures of MG from postnatal day 11 rtTA:Ascl1:GFP mice were treated with 92 molecules from a Tocris library focused on stem cell targets. Doxycycline was used to induce Ascl1 in MG for 5 days, the cells were processed by sci-Plex, and the scRNAseq output was analyzed. In vivo reprogramming of MG was performed on adult mice (Glast-CreER/LNL-tTA/tetO-Ascl1-GFP) of both sexes; Ascl1 is induced specifically in MG by tamoxifen. To induce regeneration of neurons, we make intravitreal injections of NMDA, followed by HDAC-inhibition (TSA) and hits from the in vitro screen; MG neurogenesis was assessed with immunofluorescence and scRNAseq.

Results : With the sci-Plex output, we assigned cells to clusters and assessed the effects of the treatments, focusing on compounds that increased MG-derived neurons compared to Ascl1 alone. We uncovered small molecules that target the Notch pathway (DBZ), BMP-pathway (DMH-1), TGF-B signaling (ITD-1, RepSox, SB431542), AMPK-signaling (Metformin) and the Lrc and Srk kinases (WH-4-023) that promote the ability of MG to acquire a neurogenic phenotype. Compounds from the in vitro screen were tested in vivo and both DBZ and Metformin significantly boosted neuronal regeneration in the adult mouse retina.

Conclusions : Our work outlines a strategy using scRNA-seq and plate-based screening assays to test large libraries of compounds that can enhance the ability of MG to produce neurons for cell replacement approaches.

This abstract was presented at the 2023 ARVO Annual Meeting, held in New Orleans, LA, April 23-27, 2023.

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