June 2023
Volume 64, Issue 8
Open Access
ARVO Annual Meeting Abstract  |   June 2023
Engineered bispecific antibodies as potential immune modulating therapies for uveitic glaucoma
Author Affiliations & Notes
  • Jose Quiroz
    Ophthalmology, New York Eye and Ear Infirmary of Mount Sinai, New York, New York, United States
  • Tere Williams
    Ophthalmology, SUNY Upstate Medical University Hospital, Syracuse, New York, United States
    Ophthalmology, Syracuse VA Medical Center, Syracuse, New York, United States
  • Audrey M Bernstein
    Ophthalmology, SUNY Upstate Medical University Hospital, Syracuse, New York, United States
    Ophthalmology, Syracuse VA Medical Center, Syracuse, New York, United States
  • Avnish Deobhakta
    Ophthalmology, New York Eye and Ear Infirmary of Mount Sinai, New York, New York, United States
  • Footnotes
    Commercial Relationships   Jose Quiroz None; Tere Williams None; Audrey Bernstein None; Avnish Deobhakta None
  • Footnotes
    Support  New York Eye and Ear Foundation, NIH EY030567, Merit Review Award (I01 BX005360), The Glaucoma Foundation/Research to Prevent Blindness
Investigative Ophthalmology & Visual Science June 2023, Vol.64, 5102. doi:
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    • Get Citation

      Jose Quiroz, Tere Williams, Audrey M Bernstein, Avnish Deobhakta; Engineered bispecific antibodies as potential immune modulating therapies for uveitic glaucoma. Invest. Ophthalmol. Vis. Sci. 2023;64(8):5102.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Uveitic glaucoma is a common complication in approximately 20% of patients who have uveitis. Recently, the role of immune-mediated neurodegeneration has been determined to be critical in the pathogenesis of primary open angle glaucoma.To this end, we aim to utilize single B cell sorting technology to develop immune modulating bispecific antibodies.

Methods : Peptides were synthesized for various immune surface targets and BALB/C mice were subsequently immunized with a vaccine schedule consisting of 3 immunizations at Day 0, Day 14, Day 21. Spleens from immunized mice were harvested at Day 28 for single B cell sorting to generate murine chimeric monoclonal antibodies for our various immune targets. Blood was drawn from two volunteers and PBMC isolation was performed. Human PBMCs were cultured for 5 days in the presence of apoptotic agents or media only. T-cell apoptosis was assessed by binding of Annexin V via Fluorescently Activated Cell Sorting (FACS). Percent increase of apoptosis over negative control was determined for total human PBMCs, CD4+, and CD8+ T cells. GraphPad Prism 9 software was used for statistical analysis.

Results : Spleens from CD25 peptide immunized mice demonstrated positive reactivity toward biotinylated CD25 peptide compared to unimmunized control mice spleens using FACS analysis. Incubation of agent AQZ-1 with human PBMCs for 24 hours demonstrated an increase in total PBMC apoptosis (31.3%, 25.49%), CD4+( 62.67%,75.98%),CD8+(55.33%,46.52%) at 250 nM and 125 nM respectively. Apoptosis was compared to Brefeldin A which had total human PBMC apoptosis (45.3%, 21.5%), CD4+( 28%,1.47%) CD8+(45.33%,28.33%) at 500 µM and 250 µM respectively.

Conclusions : Mice were successfully immunized with CD25 target confirmed by FACS. Single B cells were sorted and mAbs will be generated for further bispecific antibody engineering. AQZ-1 is a chimeric protein-inducer hypothesized to be an apoptosis-related immune target. AQZ-1 provided a proof-of-concept for potent apoptosis induction as compared to Brefeldin A (positive control). Combinations of immune checkpoint targets provide a viable therapeutic approach to modulate inflammation in the setting of T cell-mediated uveitis.

This abstract was presented at the 2023 ARVO Annual Meeting, held in New Orleans, LA, April 23-27, 2023.

 

Graph represents early apoptosis percent increase normalized to media only of apoptotic agents for human PBMCs, CD4+ cells, CD8+ cells. Annexin V binding was access via FACS and cell types differentiated accordingly.

Graph represents early apoptosis percent increase normalized to media only of apoptotic agents for human PBMCs, CD4+ cells, CD8+ cells. Annexin V binding was access via FACS and cell types differentiated accordingly.

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