Investigative Ophthalmology & Visual Science Cover Image for Volume 65, Issue 7
June 2024
Volume 65, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2024
Mesoporous melanin-like nanoparticles as antioxidant and drug delivery carriers for treatment of age-related macular degeneration
Author Affiliations & Notes
  • Yongsu Kwon
    Ophthalmology, The University of North Carolina at Chapel Hill School of Medicine, Chapel Hill, North Carolina, United States
  • Mary Kaufmann
    The University of North Carolina at Chapel Hill School of Medicine, Chapel Hill, North Carolina, United States
  • Julie Munsoor
    The University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, United States
  • Min Zheng
    Ophthalmology, The University of North Carolina at Chapel Hill School of Medicine, Chapel Hill, North Carolina, United States
  • Zongchao Han
    Ophthalmology, The University of North Carolina at Chapel Hill School of Medicine, Chapel Hill, North Carolina, United States
  • Footnotes
    Commercial Relationships   Yongsu Kwon None; Mary Kaufmann None; Julie Munsoor None; Min Zheng None; Zongchao Han None
  • Footnotes
    Support  BrightFocus M2022001F
Investigative Ophthalmology & Visual Science June 2024, Vol.65, 6105. doi:
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      Yongsu Kwon, Mary Kaufmann, Julie Munsoor, Min Zheng, Zongchao Han; Mesoporous melanin-like nanoparticles as antioxidant and drug delivery carriers for treatment of age-related macular degeneration. Invest. Ophthalmol. Vis. Sci. 2024;65(7):6105.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : In the face of a global aging population and the increasing prevalence of age-related macular degeneration (AMD), a condition that gradually diminishes vision, there is a demand for innovative therapeutic strategies. In this study, we have developed Indomethacin (IMC)-loaded mesoporous melanin nanoparticles (IMC- loaded mMNPs) as a drug delivery carrier for the treatment of AMD. This approach has the potential to achieve synergistic therapeutic effects by combining the antioxidant properties of melanin with the anti-inflammatory activity of IMC, thereby relieving pathological damages associated with AMD.

Methods : We introduced IMC drug onto mesoporous melanin nanoparticles (mMNPs) for its anti-inflammatory activity using EDC/NHS coupling. The size, shape, charge, and loading capacity were demonstrated through various analytical methods, including TEM, DLS, and UV-vis. We assessed the cell cytotoxicity and antioxidative properties of ICM-loaded mMNPs in an LPS-induced oxidative stress and inflammatory activity in vitro model, employing DCFDA and CCK8 assays. To evaluate the therapeutic effects of IMC-loaded mMNPs in a laser induced CNV mouse model, we performed Fundus, OCT, and immunocytochemistry analyses.

Results : The morphology of the mesoporous structure on melanin nanoparticles with uniform spherical shapes averaging approximately 148 nm was clearly revealed through TEM images. We observed that mMNPs effectively scavenged intracellular ROS production, and cell viability after LPS-induced inflammatory activity was above 80 % in the IMC-mMNPs-treated group. Furthermore, to examine the therapeutic effects in a laser-induced CNV mouse model, we injected IMC-loaded mMNPs though a single-intravitreal administration. The Fundus/OCT and DCF staining images showed that the IMC-loaded mMNPs-injected group significantly alleviated inflammatory levels and ROS generation.

Conclusions : IMC-loaded mMNPs were designed to fully utilize the advantageous properties of melanin and an anti-inflammatory drug for the treatment of pathological damages in AMD. Our findings demonstrate that IMC-loaded mMNPs can serve as an antioxidant and an anti-inflammatory agent, providing a potentially powerful treatment option for AMD patients.

This abstract was presented at the 2024 ARVO Annual Meeting, held in Seattle, WA, May 5-9, 2024.

 

A graphical illustration of mMNPs-mediated strategy to reduce oxidative stress and inflammation in a laser-induced AMD-like mouse model.

A graphical illustration of mMNPs-mediated strategy to reduce oxidative stress and inflammation in a laser-induced AMD-like mouse model.

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