Investigative Ophthalmology & Visual Science Cover Image for Volume 65, Issue 7
June 2024
Volume 65, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2024
Regulatory T cells promotes corneal epithelial wound healing through exosomal CD47
Author Affiliations & Notes
  • Chao Wei
    Eye Institute of Shandong First Medical University, Qingdao, Shandong, China
  • Yaoyao Yu
    Eye Institute of Shandong First Medical University, Qingdao, Shandong, China
  • Shengqian Dou
    Eye Institute of Shandong First Medical University, Qingdao, Shandong, China
  • Li Ma
    Eye Institute of Shandong First Medical University, Qingdao, Shandong, China
  • Weiyun Shi
    Eye Institute of Shandong First Medical University, Qingdao, Shandong, China
  • Footnotes
    Commercial Relationships   Chao Wei None; Yaoyao Yu None; Shengqian Dou None; Li Ma None; Weiyun Shi None
  • Footnotes
    Support  Shandong Provincial Key Research and Development Program (2022CXGC010505 and 2021LCZX08)
Investigative Ophthalmology & Visual Science June 2024, Vol.65, 67. doi:
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    • Get Citation

      Chao Wei, Yaoyao Yu, Shengqian Dou, Li Ma, Weiyun Shi; Regulatory T cells promotes corneal epithelial wound healing through exosomal CD47. Invest. Ophthalmol. Vis. Sci. 2024;65(7):67.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : A large body of evidence demonstrated the critical role of regulatory T cells (Tregs) in regulating wound repair and regeneration. However, the function of increased Tregs during corneal epithelial wound healing remained largely uncertain. We therefore aimed to investigate the role of Tregs in the corneal epithelial wound healing.

Methods : Based on murine corneal epithelial abrasion model, Foxp3DTR/EGFP mice and anti-CD25 antibodies were applied to deplete in vivo Tregs and evaluate their role in corneal wound healing. Single-cell RNA sequencing (scRNA seq) was performed to systemically investigate the effect of Treg on corneal limbal epithelial stem cells (LESCs) and various immunocytes. Besides, exosome released from Tregs was isolated and used to determine the potential to treat corneal wound healing under different pathological scenarios. Additionally, exosomal proteomic approach and loss-of-function experiment were employed to explore the underlying mechanism.

Results : When compared with the normal corneas, more accumulation of Tregs in the corneas during epithelial wound healing was observed. Specifical depletion of Tregs apparently impaired corneal epithelial wound closure, characterized by the lowered wound healing rate and decreased expression of Ki67 and GPHA2. Through scRNA seq analysis, some clusters of genes related with LSC function and epithelial proliferation showed a remarkably lowered expression in Treg-depleted group. By contrast, the proportions of several types of immunocytes (such as monocytes, macrophages and dendritic cells) in Treg-depleted corneas were obviously increased, the significant enrichment of inflammation-associated biological processes of the up-regulated genes in Treg-depleted corneas were also determined. More interestingly, we also found that the exosomes derived from Tregs promoted corneal epithelial wound healing, mechanistically relied on CD47, suggesting the possibility of exosome in the replace of Tregs’ function.

Conclusions : Our findings demonstrated that Tregs in corneas play vital roles in maintaining corneal homeostasis, and further highlighted Treg-derived exosomes as an alternative approach for treating corneal epithelial wound healing.

This abstract was presented at the 2024 ARVO Annual Meeting, held in Seattle, WA, May 5-9, 2024.

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