Investigative Ophthalmology & Visual Science Cover Image for Volume 65, Issue 7
June 2024
Volume 65, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2024
Measuring the kinetics of rod/cone coupling plasticity using the photovoltage of mouse cone photoreceptors
Author Affiliations & Notes
  • Nange Jin
    Vision Sciences, University of Houston, Houston, Texas, United States
  • Zhijing Zhang
    Vision Sciences, University of Houston, Houston, Texas, United States
  • Stephen C Massey
    Ophthalmology and Visual Science, The University of Texas Health Science Center at Houston, Houston, Texas, United States
  • Christophe Ribelayga
    Vision Sciences, University of Houston, Houston, Texas, United States
  • Footnotes
    Commercial Relationships   Nange Jin None; Zhijing Zhang None; Stephen Massey None; Christophe Ribelayga None
  • Footnotes
    Support  This research is funded by NIH grants: R01EY032508 (to CPR); R01EY029408 (to SCM and CPR), P30EY028102 (UTH Vision Core Grant), P30EY007551 (UH Vision Core Grant), and endowed professorship from the Foundation for Education and Research in Vision (FERV) (to CPR).
Investigative Ophthalmology & Visual Science June 2024, Vol.65, 2188. doi:
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    • Get Citation

      Nange Jin, Zhijing Zhang, Stephen C Massey, Christophe Ribelayga; Measuring the kinetics of rod/cone coupling plasticity using the photovoltage of mouse cone photoreceptors. Invest. Ophthalmol. Vis. Sci. 2024;65(7):2188.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : In mouse retina, rods and cones are electrically coupled by gap junctions made of connexin36 (Cx36). Rod/cone coupling allows light-evoked signals that originate in rods to enter cones and alter the cone membrane potential in a way that directly reflects the low threshold and slow kinetics of the rod light responses. Rod/cone coupling is not static however and is modulated by light/dark and/or circadian factors along a wide dynamic range. It remains unclear how fast these changes are implemented. We used the amount of rod signal in the cone photovoltage as a proxy to study the kinetics of rod/cone coupling increase or decrease during light/dark adaptation and/or pharmacological treatments.

Methods : Experiments were conducted on dark-adapted perfused retinal sections from wild type (WT) mice (C57BL6/J) or from mice lacking rod/cone coupling (Cx36-KO mice) (Jin et al. 2020 Science Adv. 28, aba7232). Patch-clamp recording of single cone pedicles was performed in response to series of 20-ms light flashes of increasing intensity. Light adaptation was obtained by presenting a full-field light stimulus against a dark background (500 ms in duration, 0.5 Hz) that was turned off immediately before running the intensity series every 5 min. We used the threshold of the cone photovoltage as well as the area of the slow component present at higher intensities to quantify the rod-originated signal contribution to the cone photovoltage. A reduction or increase in the rod contribution was interpreted as a decrease or an increase in rod/cone coupling, respectively.

Results : Bright light adaptation (2,000 R*/rod/500-ms, 0.5 Hz) of dark-adapted WT cones eliminated the rod component in the cone photovoltage within 10-15 min (n=5). The threshold and shape of the photovoltage of dark-adapted Cx36-KO cones were identical to those of light-adapted WT cones after 15 min (n=5). Application of the gap-junction blocker meclofenamic acid (MFA) eliminated the rod input to dark-adapted WT cones within 15 to 30 min (n=6) but had no effect on the photovoltage of dark-adapted Cx36-KO cones (n=6).

Conclusions : The data reveal the relatively slow kinetics of rod/cone decoupling that take tens of minutes to fully develop. Experiments are underway to determine the kinetics of the dark- and/or pharmacologically induced increase in rod/cone coupling.

This abstract was presented at the 2024 ARVO Annual Meeting, held in Seattle, WA, May 5-9, 2024.

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